Results 101 to 110 of about 25,202 (233)
Immunohistochemical Analysis of Matrix Metalloproteinases-1,-9 and Tenascin in Odontogenic Lesions
European Journal of Inflammation, 2014 Ameloblastoma (ABL) and keratocystic odontogenic tumor (KOT) are benign odontogenic tumors with local aggressive behaviors. The purpose of our study was to compare MMP-1, MMP-9 and tenascin staining patterns between “aggressive” ameloblastoma ...
E. Bariş +3 more
doaj +1 more source
Comprehensive Physiology, Volume 16, Issue 6, December 2026.Schematic diagram illustrating the potential mechanisms and core molecules contributing to the development of PE via mediating pyroptosis: focus on trophoblasts and endothelial cells. Pyroptosis of trophoblast cells in the placenta of preeclampsia not only inhibits its invasion, migration, and differentiation, causing local infiltration and ...
Xianyi Zhou +5 more
wiley +1 more source
Growth promoting signaling by tenascin-C [corrected].
, 2004 Tenascin-C is an adhesion-modulating extracellular matrix molecule that is highly expressed in tumor stroma and stimulates tumor cell proliferation. Adhesion of T98G glioblastoma cells to a fibronectin substratum is inhibited by tenascin-C.
Ruiz, C. +17 more
core +1 more source
Glia, Volume 74, Issue 12, December 2026.Brain aging alters perineuronal nets and neuroinflammation. 4‐methylumbelliferone reversed these aging‐associated changes. 4‐methylumbelliferone reduced age‐associated cognitive decline. Six months of chronic oral treatment was well tolerated. ABSTRACT Aging‐associated cognitive decline is closely linked to dysregulated glial activity and chronic ...
Anda Cimpean +7 more
wiley +1 more source
Expression of ECM-Tenascin in Ethylnitrosourea-Induced Rat Glioma [PDF]
, 1995 Tenascin (TN) is a major extracellular matrix glycoprotein that shows a restricted distribution during fetal organogenesis and is also involved in tumor proliferation and invasion.
Wu, Zhiren
core
The regulation of tenascin expression by tissue microenvironments
, 2009 Tenascins are a family of four extracellular matrix proteins: tenascin-C, X, R and W. The four members of the family have strikingly diverse patterns of expression during development and in the adult organism indicating independent mechanisms of ...
Richard P. Tucker +5 more
core +1 more source
ASN Neuro, 2010 The heparan sulfate proteoglycan glypican-1, the chondroitin sulfate proteoglycan phosphacan/RPTP (receptor protein-tyrosine phosphatase)-ζ/β and the extracellular matrix protein tenascin-C were all found to be expressed by neural stem cells and by ...
Mary Abaskharoun +3 more
doaj +1 more source
Regulation of IL‐13 Signaling by Retinoic Acid in Airway Epithelial Cells
The FASEB Journal, Volume 40, Issue 21, 15 November 2026.Our proteomics and subsequent biochemical analyses reveal the effects of retinoic acid receptor (RAR) agonists, ATRA (pan‐RAR agonist), or CD1530 (RARγ agonist) in counteracting molecular changes induced by IL‐13 relevant to airway inflammation and remodeling, such as proteins involved in epithelial‐to‐mesenchymal transition (EMT) and extracellular ...
Vanshika Patel +5 more
wiley +1 more source
Animal, 2016 The aim of this study was to highlight the relationships between decorin, tenascin-X and type XIV collagen, three minor molecules of extracellular matrix (ECM), with some structural parameters of connective tissue and its content in total collagen, its ...
A. Dubost +3 more
doaj +1 more source
Further Insights Into the Mechanisms of Lymph Node Metastasis
Cancer Medicine, Volume 15, Issue 11, November 2026.ABSTRACT Metastasis of malignant tumors arising from epithelial organs to sentinel lymph nodes has important implications for prognosis, staging, and therapeutic decision‐making. Recent insights into anatomic pathways, pathogenesis, molecular interactions, and genetic factors have encouraged scientists and clinicians to develop drugs that target ...
S. David Nathanson +5 more
wiley +1 more source