Results 171 to 180 of about 1,153,662 (221)
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ANTITHROMBOTIC, ANTICOAGULANT, AND THROMBOLYTIC AGENTS
Dental Clinics of North America, 1996There are a growing number of clinical indications for the use of antithrombotic, anticoagulant, and thrombolytic agents. Clinicians must recognize that these patients require multidisciplinary treatment and coordination of care, and an assessment of the patient's level of anticoagulation is imperative to ensure values that may preclude problematic ...
G T, Terezhalmy, A E, Lichtin
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Pharmacologic Review of Thrombolytic Agents
Critical Care Nursing Clinics of North America, 1990Thrombolytic therapy has significantly reduced the morbidity and mortality that was once associated with acute myocardial infarction. Because of the substantial benefits associated with this therapy, investigation has intensified in search of the optimal agent or agents.
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Comparative safety of thrombolytic agents
The American Journal of Cardiology, 1991Three available thrombolytic agents, streptokinase, alteplase, and anistreplase, have been shown to have similar effects on preservation of left ventricular function and mortality reduction after acute myocardial infarction (AMI). The agents are, however, quite different with respect to their safety profiles.
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Nihon rinsho. Japanese journal of clinical medicine, 2014
Thrombolytic agents positively resolve existing thrombi by accelerating the activity of plasmin, a key enzyme of the fibrinolytic pathway. The main drug in use is a plasminogen activator (PA) which degrades plasminogen to plasmin. PA is classified into urokinase-type (u-PA) and tissue-type (t-PA).
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Thrombolytic agents positively resolve existing thrombi by accelerating the activity of plasmin, a key enzyme of the fibrinolytic pathway. The main drug in use is a plasminogen activator (PA) which degrades plasminogen to plasmin. PA is classified into urokinase-type (u-PA) and tissue-type (t-PA).
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Comparison of thrombolytic agents
Critical Care Nursing Quarterly, 1989The new generation of plasminogen activators promises certain advantages over the first-generation agents, particularly enhanced clot specificity. The tissue distribution, half-life, therapeutic profile, and optimum dosage, however, need to be evaluated for each chemically different agent.
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Thrombolytic, antiplatelet, and antithrombotic agents
The American Journal of Cardiology, 1992The relative efficacy and safety of individual thrombolytic agents, administered alone and with antiplatelet and antithrombotic drugs, in the treatment of acute myocardial infarction are presented. The clinical benefits and risks of treatment choices are discussed in relation to the mechanisms of the formation and prevention of thrombus and ...
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Nihon Geka Gakkai zasshi, 1984
The clinical efficacy of thrombolytic agents, urokinase, lysyl-plasminogen and batroxobin, was evaluated on the patients with deep venous thrombosis. In the group treated with combination of batroxobin, lysyl-plasminogen and urokinase, significantly better efficacy was obtained over the urokinase group in terms of judgement on venography and clinical ...
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The clinical efficacy of thrombolytic agents, urokinase, lysyl-plasminogen and batroxobin, was evaluated on the patients with deep venous thrombosis. In the group treated with combination of batroxobin, lysyl-plasminogen and urokinase, significantly better efficacy was obtained over the urokinase group in terms of judgement on venography and clinical ...
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Thrombolytic Agents in Critical Care
Critical Care Nursing Clinics of North America, 1993Thrombolytic therapy, when instituted early in optimum dosage, has had an important role in improving the survival of critically ill patients. Thrombolysis is but one component of the processes associated with hemostasis, the mechanism by which humans are protected from death by hemorrhage following injury to a blood vessel.
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