Results 171 to 180 of about 8,086 (190)

Binding of TMPRSS2-ERG to BAF Chromatin Remodeling Complexes Mediates Prostate Oncogenesis. [PDF]

Mol Cell, 2018
Sandoval GJ, Pulice JL, Pakula H, Schenone M, Takeda DY, Pop M, Boulay G, Williamson KE, McBride MJ, Pan J, St Pierre R, Hartman E, Garraway LA, Carr SA, Rivera MN, Li Z, Ronco L, Hahn WC, Kadoch C.   +18 more
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A Prospective Study of Aspirin Use and Prostate Cancer Risk by TMPRSS2:ERG Status. [PDF]

Cancer Epidemiol Biomarkers Prev, 2018
Stopsack KH, Gonzalez-Feliciano AG, Peisch SF, Downer MK, Gage RA, Finn S, Lis RT, Graff RE, Pettersson A, Pernar CH, Loda M, Kantoff PW, Ahearn TU, Mucci LA, Transdisciplinary Prostate Cancer Partnership (ToPCaP).   +14 more
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RNA-mediated TMPRSS2-ERG gene fusion in prostate cancer

, 2020
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FOXA1 Alterations in Prostate Cancer: Expression, Mutation Classes, and Copy Number Changes. [PDF]

In Vivo
Lehrer S, Rheinstein P.
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Characterize the difference between TMPRSS2-ERG and non-TMPRSS2-ERG fusion patients by clinical and biological characteristics in prostate cancer

Gene, 2018
The TMPRSS2-ERG gene fusion were frequently found in prostate cancer, and thought to play some fundamental mechanisms for the development of prostate cancer. However, until now, the clinical and prognostic significance of TMPRSS2-ERG gene fusion was not fully understood.
Shiyuan, Wang, Qi, Zhang, Dandan, Xu, Yi, Pan, Yingli, Lv, Xiaowen, Chen, Yongchun, Zuo, Lei, Yang   +7 more
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Atypical Adenomatous Hyperplasia of Prostate Lacks TMPRSS2-ERG Gene Fusion

American Journal of Surgical Pathology, 2013
Atypical adenomatous hyperplasia (AAH) is a distinct entity in prostate pathology, defined as a well-circumscribed lobule of closely packed crowded small glands or acini. Although it has been proposed as a precursor lesion to prostate cancer, the biological nature of AAH is currently uncertain.
Liang, Cheng, Darrell D, Davidson, Gregory T, Maclennan, Antonio, Lopez-Beltran, Rodolfo, Montironi, Mingsheng, Wang, Puay-Hoon, Tan, Lee Ann, Baldridge, Shaobo, Zhang   +8 more
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Protocols for Studies on TMPRSS2/ERG in Prostate Cancer

2018
TMPRSS2/ERG is the most common type of gene fusions found in human prostate cancer. There are two important features of TMPRSS2/ERG fusions. One is that these gene fusions lead to ectopic expression of ERG, an ETS family transcription factor, in prostate epithelial cells from the 5' control region of an androgen/estrogen dual-responsive gene, TMPRSS2 ...
Hubert, Pakula, Douglas E, Linn, Daniel R, Schmidt, Marit, Van Gorsel, Matthew G, Vander Heiden, Zhe, Li   +5 more
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TMPRSS2-ERG and PTEN loss in prostate cancer

Nature Genetics, 2009
Two studies show that the common recurrent gene fusion between TMPRSS2 and ERG promotes prostate cancer in both mouse and humans when PTEN is concurrently lost. In human prostate cancer, the presence of both these aberrations may be indicative of poor prognosis, suggesting that preclinical therapeutic research should target both of these pathways.
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Precision prevention of TMPRSS2:ERG prostate cancer.

Journal of Clinical Oncology, 2016
78 Background: Increased integration of tumor biomarker data into prostate cancer epidemiology studies is needed to identify molecular subtypes that underlie its etiology and progression. We hypothesize that the TMPRSS2:ERG gene fusion is a unique prostate cancer subtype that is etiologically distinct from cancers lacking TMPRSS2:ERG.
Lorelei A. Mucci, Thomas Ahearn, Kathryn Penney, Andreas Pettersson, Rebecca E Graff, Philip W. Kantoff, Stephen Finn, Massimo Loda   +7 more
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TMPRSS2-ERG , a Mover and a Shaker

Science Translational Medicine, 2010
TMPRSS2-ERG modulates critical pathways to promote prostate cancer progression.
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