Results 231 to 240 of about 230,179 (340)

Influence of the Vaginal Microbiota on Toxic Shock Syndrome Toxin 1 Production by Staphylococcus aureus

open access: yesApplied and Environmental Microbiology, 2013
Roderick A. MacPhee   +6 more
semanticscholar   +1 more source

Primary Streptococcal Peritonitis in a Postpartum Patient: A Rare but Life‐Threatening Condition

open access: yesClinical Case Reports, Volume 14, Issue 3, March 2026.
ABSTRACT We present the case of a 43‐year‐old postpartum woman who developed primary streptococcal peritonitis with a fulminant septic course requiring intensive care. Initial symptoms were nonspecific and improved temporarily, but the patient returned the next day with recurrent pain and markedly elevated inflammatory markers.
Matthias Pimiskern, Helmut Trimmel
wiley   +1 more source

Severe Metformin‐Associated Lactic Acidosis—A Case Report

open access: yesClinical Case Reports, Volume 14, Issue 3, March 2026.
ABSTRACT Metformin, a widely prescribed and generally safe drug for diabetes mellitus, can rarely lead to metformin‐associated lactic acidosis—a severe, potentially fatal complication. Early recognition, emergency hemodialysis, and supportive management are crucial for favorable outcomes, highlighting the importance of prompt diagnosis and intervention
Elizabet Artinyan   +7 more
wiley   +1 more source

Skin levels of toxic shock syndrome toxin-1 predict the severity of atopic dermatitis. [PDF]

open access: yesJ Allergy Clin Immunol
Woods B   +9 more
europepmc   +1 more source

Successful Management of Suspected Epstein–Barr Virus‐Associated Hemophagocytic Lymphohistiocytosis in BRAF‐Mutant Cholangiocarcinoma Following Treatment With Immune Checkpoint Inhibitors and BRAF/MEK Inhibitors: A Case Report

open access: yesCancer Reports, Volume 9, Issue 3, March 2026.
ABSTRACT Background The BRAF V600E mutation is a rare genetic alteration in cholangiocarcinoma for which sequential therapy with immune checkpoint inhibitors (ICIs) and BRAF/MEK inhibitors may be effective. However, the full spectrum of adverse events associated with the sequential use of these agents remains unclear. Hemophagocytic lymphohistiocytosis
Akira Shibata   +9 more
wiley   +1 more source

Ninjurin1 in cardiovascular and vascular biology: From molecular mechanisms to therapeutic opportunities

open access: yesClinical and Translational Medicine, Volume 16, Issue 3, March 2026.
NINJ1 mediates PMR in cardiovascular cell death, releasing DAMPs that amplify vascular inflammation. Dual NINJ1 paradox: membrane‐bound form drives endothelial NF‐κB/CXCL‐8 inflammation while soluble form provides atheroprotection. Pericyte NINJ1 regulates the critical balance between angiogenic sprouting and vessel maturation in ischaemic tissues ...
Muhammad Mamunur Rashid Mahib
wiley   +1 more source

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