Results 191 to 200 of about 3,598,857 (362)
A novel microphysiological system that can mimic sterile or non‐sterile injury demonstrates that macrophage‐derived extracellular vesicles modulate neutrophil migratory responses to these different wound states. The model showed that resolution of inflammation through neutrophil reverse migration is driven by IL‐8 derived from extracellular vesicles ...
Kehinde Adebayo Babatunde+5 more
wiley +1 more source
Multicast reliable traffic engineering technique for SDN-Fog based IoUT. [PDF]
Mohammadi R.
europepmc +1 more source
A Survey on the Contributions of Software-Defined Networking to Traffic Engineering
A. Mendiola+3 more
semanticscholar +1 more source
The elasticity of lipid nanoparticles (LNPs) is essential to their tumor‐targeting efficacy. Softer LNPs penetrate tumors faster and more easily enter into softer cancer cells, while stiffer LNPs show prolonged retention in stiffer tumors. The presence of protumoral M2 macrophages enhances the LNPs uptake into stiffer cancer cells, facilitating ...
Eunhee Lee+5 more
wiley +1 more source
OPTIMAL DECENTRALIZED ALGORITHMS FOR TRAFFIC ENGINEERING IN THE INTERNET
B.A. Movsichoff, Constantino Lagoa
openalex +1 more source
The release of COL6A3 by fibroblasts is sensed by the CD44 receptor on melanocytes, activating glutathione (GSH) metabolism and increasing melanocyte survival during skin organoid culture. SEMA3C released by bulge cells binds to the NRP1 receptor on melanocytes, regulating microtubule stability and promoting melanocyte recruitment and function during ...
Tingting Li+17 more
wiley +1 more source
MPLS Multicast Traffic Engineering
In this paper, we present multicast traffic engineering and we compare it to unicast traffic engineering. We study the advantages given by the integration of multicasting and MPLS. We present current proposals for multicast traffic engineering and using MPLS network. We describe our approach, the MPLS multicast tree (MMT) protocol.
Boudani, Ali+2 more
openaire +1 more source
Cisplatin causes reactive oxygen species accumulation, leading to apoptosis and inflammation in cochlear hair cells. Small extracellular vesicles primarily derived from the damaged hair cells likely contribute to cisplatin‐induced ototoxicity, carrying a variety of microRNAs and proteins.
Jingru Ai+14 more
wiley +1 more source