Results 241 to 250 of about 570,549 (333)
Editorial: Rehabilitation interventions for mild traumatic brain injury. [PDF]
Anwar F.
europepmc +1 more source
DNA‐based hydrogels serve dual roles as structural scaffolds and therapeutic platforms, offering innovative solutions for musculoskeletal regeneration with multifunctional abilities. Abstract Musculoskeletal disorders, including bone fractures, osteoarthritis, and muscle injuries, represent a leading cause of global disability, revealing the urgency ...
Ruijianghan Shi+4 more
wiley +1 more source
Reconsidering the symmetric assumption in CPP-PRx modeling for traumatic brain injury. [PDF]
Zhang H, Zhou W, Ding Y.
europepmc +1 more source
Prospective memory retraining in adults with traumatic brain injury
Roseann Hannon
openalex +1 more source
By drawing inspiration from biological neural hierarchies and the working mechanisms of plasticity, researchers have constructed a series of bionic devices, including sensory devices, synapse devices, and artificial neural systems. They committed to simulating and surpassing the biological information processing function, thereby realizing the ...
Lu Yang+5 more
wiley +1 more source
Traumatic Brain Injury and Coenzyme Q10: An Overview. [PDF]
Mantle D+3 more
europepmc +1 more source
Parabiosis, Assembloids, Organoids (PAO)
This review evaluates parabiosis, organoids, and assembloids as complementary disease models spanning systemic, organ, and multi‐organ levels. It highlights their construction strategies, applications, and current limitations, while emphasizing their integration with frontier technologies such as artificial intelligence, organ‐on‐a‐chip, CRISPR, and ...
Yang Hong+5 more
wiley +1 more source
EFFECTS OF AEROBIC CIRCUIT TRAINING IN ADULTS WITH TRAUMATIC BRAIN INJURY
Gary Rowland, Yagesh Bhambhani
openalex +1 more source
Aging involves physiological decline and heightened disease susceptibility, necessitating effective interventions. Redox‐responsive selenium‐loaded nanoparticles (SeMSNs) are developed, which delay cellular senescence by reducing oxidative stress and senescence markers (p16/p21). In aged mice, SeMSNs extend lifespan, improve organ function, and restore
Yang Yu+15 more
wiley +1 more source