Results 41 to 50 of about 583,291 (307)

Analysis of influencing factors of acute kidney injury after spontaneous intracerebral hemorrhage surgery

open access: yesChinese Journal of Contemporary Neurology and Neurosurgery
Objective To analyze the influencing factors of acute kidney injury (AKI) after surgery in spontaneous intracranial hemorrhage (sICH). Methods A total of 151 patients with sICH who underwent surgery in Tianjin Huanhu Hospital from March 2023 to June ...
ZHANG Lei   +4 more
doaj   +1 more source

Pathophysiology of traumatic brain injury.

open access: yesBritish Journal of Anaesthesia, 2007
The knowledge of the pathophysiology after traumatic head injury is necessary for adequate and patient-oriented treatment. As the primary insult, which represents the direct mechanical damage, cannot be therapeutically influenced, target of the treatment
C. Werner, K. Engelhard
semanticscholar   +1 more source

Mild traumatic brain injury increases engagement in criminal behaviour 10 years later: a case–control study

open access: yesFrontiers in Psychiatry, 2023
IntroductionSustaining a mild traumatic brain injury (mTBI) has been linked to increased criminal behaviour in later life. However, previous studies have not controlled for the number of injuries, gender, social deprivation, impact of past behaviour, or ...
Alice Theadom   +4 more
doaj   +1 more source

Animal models of traumatic brain injury

open access: yesNature Reviews Neuroscience, 2013
Traumatic brain injury (TBI) is a leading cause of mortality and morbidity both in civilian life and on the battlefield worldwide. Survivors of TBI frequently experience long-term disabling changes in cognition, sensorimotor function and personality ...
Y. Xiong, A. Mahmood, M. Chopp
semanticscholar   +1 more source

Cellular infiltration in traumatic brain injury

open access: yesJournal of Neuroinflammation, 2020
Traumatic brain injury leads to cellular damage which in turn results in the rapid release of damage-associated molecular patterns (DAMPs) that prompt resident cells to release cytokines and chemokines.
Aftab Alam   +6 more
semanticscholar   +1 more source

An Objective Assessment of Neuromotor Control Using a Smartphone App After Repeated Subconcussive Blast Exposure

open access: yesSensors
Subconcussive blast exposure has been shown to alter neurological functioning. However, the extent to which neurological dysfunction persists after blast exposure is unknown.
Charlend K. Howard   +11 more
doaj   +1 more source

Neuroimmunology of Traumatic Brain Injury: Time for a Paradigm Shift

open access: yesNeuron, 2017
SUMMARY Traumatic brain injury (TBI) is a leading cause of morbidity and disability, with a considerable socioeconomic burden. Heterogeneity of pathoanatomical subtypes and diversity in the pathogenesis and extent of injury contribute to differences in ...
Yasir N. Jassam   +4 more
semanticscholar   +1 more source

Change in Mental Health and Resilience in Childhood Cancer Survivors After Attending a Person‐Centred State‐of‐the‐Art Late Effects Clinic—on Behalf of the PanCareFollowUp Consortium

open access: yesPediatric Blood &Cancer, EarlyView.
ABSTRACT Background Person‐centred follow‐up care based on evidence‐based clinical practice guidelines and providing individualised information should help to inform and reassure survivors about their medical and psychosocial situation and provide treatment and support where needed.
Gisela Michel   +36 more
wiley   +1 more source

A randomized controlled trial protocol for people with traumatic brain injury enrolled in a healthy lifestyle program (GLB-TBI)

open access: yesContemporary Clinical Trials Communications, 2019
Weight gain is prevalent among people with traumatic brain injury (TBI) and may be attributable to environmental or injury-specific factors such as mobility impairment, endocrine dysfunction, behavioral and emotional disorders, and sensory loss.
Simon Driver   +5 more
doaj   +1 more source

The therapeutic opportunities and pitfalls of using iron ion chelators to treat neurodegenerative diseases

open access: yesFEBS Letters, EarlyView.
Although too much iron in the brain promotes neurodegeneration, iron ion chelators have had mixed effects in clinical trials. This review explains why; some chelators do not render the iron redox‐inactive (e.g., L1) whereas others do (e.g., desferrioxamine).
Barry Halliwell
wiley   +1 more source

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