Results 181 to 190 of about 8,387 (243)

Yixinjiedu Formula Attenuates Pressure Overload-Induced Cardiac Dysfunction by Suppressing Ferroptosis and Restoring Mitophagy via the PINK1/Parkin Axis. [PDF]

open access: yesPharmaceuticals (Basel)
Xie K   +11 more
europepmc   +1 more source

[The action of trimetazidine (TMZ)].

open access: yesWiadomosci lekarskie (Warsaw, Poland : 1960), 1988
T, Petelenz   +2 more
openaire   +1 more source

Trimetazidine

open access: yesReactions Weekly, 2015
Coronary revascularization is not a suitable option for all patients with chronic stable angina (CSA) and, even when such a strategy is adopted, in conjunction with optimal medical therapy (OMT) as suggested by international guidelines, not all of them succeed to be symptom free.
HUQI, ALDA   +3 more
core   +3 more sources

The effect of trimetazidine in the treatment of microvascular angina

open access: yesInternational Journal of Angiology, 1999
Although the pathophysiology of microvascular angina is unclear, intracellular metabolic changes are believed to be the main factors. Trimetazidine has an intracellular metabolic effect in coronary insufficiency. The effect of trimetazidine in microvascular angina is unknown.
Nalbantgil S.   +4 more
openaire   +3 more sources
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Cardioprotective effects of trimetazidine: a review

Current Medical Research and Opinion, 2003
The efficacy of trimetazidine, an anti-ischaemic agent, has been largely assessed and presented in the international literature through its metabolic effects, selective and specific fatty acid oxidation inhibition and lack of haemodynamic effects in stable angina pectoris. As such, trimetazidine has opened up a new class of metabolic agents that reduce
exaly   +3 more sources

Trimetazidine

Drugs, 2012
K J McClellan, G L Plosker
exaly   +2 more sources

Trimetazidine, a Metabolic Modulator, Has Cardiac and Extracardiac Benefits in Idiopathic Dilated Cardiomyopathy

open access: yesCirculation, 2008
BACKGROUND: The anti-ischemic agent trimetazidine improves ejection fraction in heart failure that is hypothetically linked to inhibitory effects on cardiac free fatty acid (FFA) oxidation. However, FFA oxidation remains unmeasured in humans.
Kjell Någren   +2 more
exaly   +4 more sources

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