Results 181 to 190 of about 8,387 (243)
Yixinjiedu Formula Attenuates Pressure Overload-Induced Cardiac Dysfunction by Suppressing Ferroptosis and Restoring Mitophagy via the PINK1/Parkin Axis. [PDF]
Xie K +11 more
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A translational mechanistic synthesis of ischemia-reperfusion injury in experimental flap models toward free flap salvage. [PDF]
Akdemir O.
europepmc +1 more source
Emerging Pharmacological Strategies for Cardiac Amyloidosis: A Qualitative Analysis of Interventional Clinical Trials Registered on ClinicalTrials.Gov. [PDF]
Harbi MH, Alzahrani YA.
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[The action of trimetazidine (TMZ)].
T, Petelenz +2 more
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Coronary revascularization is not a suitable option for all patients with chronic stable angina (CSA) and, even when such a strategy is adopted, in conjunction with optimal medical therapy (OMT) as suggested by international guidelines, not all of them succeed to be symptom free.
HUQI, ALDA +3 more
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The effect of trimetazidine in the treatment of microvascular angina
Although the pathophysiology of microvascular angina is unclear, intracellular metabolic changes are believed to be the main factors. Trimetazidine has an intracellular metabolic effect in coronary insufficiency. The effect of trimetazidine in microvascular angina is unknown.
Nalbantgil S. +4 more
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Cardioprotective effects of trimetazidine: a review
Current Medical Research and Opinion, 2003The efficacy of trimetazidine, an anti-ischaemic agent, has been largely assessed and presented in the international literature through its metabolic effects, selective and specific fatty acid oxidation inhibition and lack of haemodynamic effects in stable angina pectoris. As such, trimetazidine has opened up a new class of metabolic agents that reduce
exaly +3 more sources
BACKGROUND: The anti-ischemic agent trimetazidine improves ejection fraction in heart failure that is hypothetically linked to inhibitory effects on cardiac free fatty acid (FFA) oxidation. However, FFA oxidation remains unmeasured in humans.
Kjell Någren +2 more
exaly +4 more sources

