Results 141 to 150 of about 6,764,388 (368)
Correction to: Macrotroponin Complex as a Cause for Cardiac Troponin Increase after COVID-19 Vaccination and Infection [PDF]
openalex +1 more source
Abstract Aims The extent of irreversible cardiomyocyte necrosis after acute myocardial infarction (AMI) is a major determinant of residual left ventricular (LV) function and clinical outcome. Cell therapy based on CD34+ cells has emerged as an option to help repair the myocardium and to improve outcomes.
Jerome Roncalli +17 more
wiley +1 more source
Cardiovascular (CV) disease remains the leading cause of morbidity and mortality worldwide, highlighting the necessity of understanding its underlying molecular and pathophysiological pathways.
Mihail Celeski +11 more
doaj +1 more source
Evaluation of cardiac markers and some serumic biochemical parameters in seropositive horses with equine influenza [PDF]
This study was performed to evaluate the effect of influenza in horses on the electrocardiogram changes, cardiac arrhythmias and concentration of cardiac troponin in serum and serumic activity of some enzymes.
ali hassanpour +2 more
doaj
Calcium-Induced Conformational Changes in the Cardiac Isoform of the Troponin Complex Monitored By Hydrogen/deuterium Exchange and Fourier Transform Ion Cyclotron Resonance Mass Spectrometry [PDF]
George M. Bou-Assaf +4 more
openalex +1 more source
Abstract Aims Immune checkpoint inhibitors (ICI) are the cornerstone of modern oncology; however, side effects such as ICI‐related myocarditis (irM) can be fatal. Recently, Bonaca proposed criteria for irM; however, it is unknown if they correlate well with cardiovascular (CV) ICI‐related adverse events.
Lorenzo Braghieri +12 more
wiley +1 more source
A Central Role for Troponin C Amino-Terminal α-Helix in Vertebrate Thin Filament Ca2+-Activation
Troponin C (TnC) is the Ca2+-sensing subunit of troponin that is responsible for activating thin filaments in striated muscle, and, in turn, for regulating the systolic and diastolic contractile function of cardiac muscle.
Yun Shi +8 more
doaj +1 more source
Abstract Aims Myocardial inflammation and impaired mitochondrial oxidative capacity are hallmarks of heart failure (HF) pathophysiology. The extent of myocardial inflammation in patients suffering from ischaemic cardiomyopathy (ICM) or dilated cardiomyopathy (DCM) and its association with mitochondrial energy metabolism are unknown.
Julius Borger +15 more
wiley +1 more source
The effect of heart and skeletal muscle troponin complexes and calmodulin on the Ca2+‐dependent reactions of phosphorylase kinase isoenzymes [PDF]
K Yoshikawa +4 more
openalex +1 more source
Considerations for drug trials in hypertrophic cardiomyopathy
Abstract Hypertrophic cardiomyopathy (HCM) is a heterogeneous condition with potentially serious manifestations. Management has traditionally comprised therapies to palliate symptoms and implantable cardioverter‐defibrillators to prevent sudden cardiac death. The need for disease‐modifying therapies has been recognized for decades.
John P. Farrant +17 more
wiley +1 more source

