Results 201 to 210 of about 182,452 (358)
Multisectoral approaches to the prevention and control of vector-borne diseases: lessons learned from case studies. [PDF]
Nunes JB +8 more
europepmc +1 more source
In hypoxic microenvironment, WNT5A is predominantly secreted by tumor‐associated macrophages. Hypoxia‐induced WTAP mediates ROR1 stability by m6A modifications in a HuR‐dependent manner in Glioma stem cells (GSCs). WNT5A activates the WNT pathway via ROR1 binding on GSCs, driving glioma‐derived endothelial cells (GDECs) differentiation.
Xiaoyong Chen +15 more
wiley +1 more source
Comparative analysis of diagnostic methods and genetic analysis for canine vector-borne diseases in Thailand. [PDF]
Junsiri W, Taweethavonsawat P.
europepmc +1 more source
Genetic manipulation of vectors: A potential novel approach for control of vector-borne diseases
Barry J. Beaty
openalex +2 more sources
Glutamine deprivation triggers transient DNA damage yet activates adaptive repair in hepatocellular carcinoma cells. We identify TRIB3 as a stress‐induced nuclear scaffold that associates with DDX5 and G‐quadruplex DNA atBRCA1 andRAD51AP1 promoters. TRIB3 loss increases G4 accumulation, suppresses HR gene transcription, elevates γ‐H2A.X, and sensitizes
Qiang Ji +10 more
wiley +1 more source
Immunology of vector-borne diseases: the role of immunopharmacology in controlling viral and parasitic infections. [PDF]
Abbasi E.
europepmc +1 more source
Reported Vector-Borne Diseases — China, 2018
Qiyong, Liu, Yuan, Gao
openaire +2 more sources
Osteosarcoma stemness is driven by the ITGB2‐COPS3‐SOX2 signaling axis. This study reveals that nuclear COPS3 stabilizes SOX2, which in turn undergoes liquid‐liquid phase separation to promote stemness. Based on this mechanism, a novel COPS3 inhibitor, Z‐5891, was developed, effectively suppressing tumor growth and stemness in vivo, offering a ...
Lei Guo +7 more
wiley +1 more source
Developing and validating modular surveys for vector-borne diseases: A study protocol. [PDF]
Finucane S +4 more
europepmc +1 more source

