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Anesthetic cardioprotection in coronary artery surgery? The results of the Volatile Anesthetics and Cardioprotection multicentre ANalysis (VACMAN) [PDF]

, 2009
De Hert, Stefaan, HUYS, JEROEN, Vacman Study Group,, Wouters, Patrick   +3 more
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Cardioprotection by volatile anesthetics

Vascular Pharmacology, 2005
Preconditioning describes a very powerful endogenous mechanism by which the heart may be protected against ischemia and reperfusion injury. Transient administration of a volatile anesthetic before a prolonged ischemic episode reduces myocardial infarct size to a degree comparable to that observed during ischemic preconditioning.
Martin W, Bienengraeber, Dorothee, Weihrauch, Judy R, Kersten, Paul S, Pagel, David C, Warltier   +4 more
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Volatile anesthetic-induced preconditioning

Perfusion, 2013
The myocardium has an innate ability to protect itself from ischemic events. This protection occurs when the myocardium is exposed to a brief ischemic period prior to a more extreme ischemic event. This is termed ischemic preconditioning. Ischemic preconditioning induces a series of molecular pathways that protect the cardiac myocyte; first, for a ...
T, Swyers, D, Redford, D F, Larson
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Mutagenicity of Volatile Anesthetics

Anesthesiology, 1976
The mutagenicity of halothane was tested in an in-vitro microbial assay system employing two histidine-dependent mutants of Salmonella typhimurium, TA98 and TA100, Halothane in concentrations ranging from 0.1 to 30 per cent was incubated with bacteria in the presence or absence of a metabolic activation system prepared from either rat liver treated ...
J M, Baden, M, Brinkenhoff, R S, Wharton, B A, Hitt, V F, Simmon, R I, Mazze   +5 more
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Volatile anesthetic-induced cardiac preconditioning

Journal of Anesthesia, 2007
Pharmacological preconditioning with volatile anesthetics, or anesthetic-induced preconditioning (APC), is a phenomenon whereby a brief exposure to volatile anesthetic agents protects the heart from the potentially fatal consequences of a subsequent prolonged period of myocardial ischemia and reperfusion.
Stadnicka, Anna, Marinović, Jasna, Ljubković, Marko, Bienengraeber, Martin W., Bošnjak, Željko J.   +4 more
openaire   +3 more sources

Volatile Anesthetics and Cardiac Function

Seminars in Cardiothoracic and Vascular Anesthesia, 2006
All volatile anesthetics have been shown to induce a dose-dependent decrease in myocardial contractility and cardiac loading conditions. These depressant effects decrease myocardial oxygen demand and may, therefore, have a beneficial role on the myocardial oxygen balance during myocardial ischemia.
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[Volatile anesthetics].

Der Anaesthesist, 2004
None of the currently available inhaled anesthetics has all of the properties of an "ideal" inhaled agent. The exceptionally low solubility of desflurane and sevoflurane offers a significantly greater precision of control over maintenance of anesthesia and a potential for a more rapid recovery from anesthesia than other inhaled anesthetics. Sevoflurane
M, Loscar, P, Conzen
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Drug Interactions: Volatile Anesthetics and Opioids

Journal of Clinical Anesthesia, 1997
Multiple drugs are used to provide anesthesia. Volatile anesthetics are commonly combined with opioids. Several studies have demonstrated that small doses of opioid (i.e., within the analgesic range) result in a marked reduction in minimum alveolar concentration (MAC) of the volatile anesthetic that will prevent purposeful movement in 50% of patients ...
P S, Glass, T J, Gan, S, Howell, B, Ginsberg   +3 more
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Volatile Anesthetics Depress Spinal Motor Neurons

Anesthesiology, 1996
Background Depression of spinal alpha-motor neurons apparently plays a role in the surgical immobility induced by isoflurane. Using the noninvasive technique of F-wave analysis, the authors tested the hypothesis that depressed motor neuron excitability is an effect common to other clinically relevant inhaled anesthetics.
I J, Rampil, B S, King
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Volatile Anesthetics and Neuroprotection

2015
Since 1963, many studies have demonstrated the protective and preconditioning effects of volatile anesthetics on cerebral ischemia, and it has also been suggested that they can delay neuronal cell death, especially in the developing brain. Various molecular mechanisms involving the numerous pathways of the neuronal cell death cascade have been reported
Yasunori Mishima, Kazuo Ushijima
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