Results 71 to 80 of about 39,734 (217)

Yersinia pestis intracellular parasitism of macrophages from hosts exhibiting high and low severity of plague. [PDF]

open access: yesPLoS ONE, 2012
BACKGROUND: Yersinia pestis causes severe disease in natural rodent hosts, but mild to inapparent disease in certain rodent predators such as dogs. Y.
Duraisamy Ponnusamy   +1 more
doaj   +1 more source

De novo or Salvage? Nucleotide Availability as a Driver of Bacterial Adaptation and Virulence

open access: yesMicrobiologyOpen, Volume 15, Issue 4, August 2026.
Bacterial pathogens survive in different host environments by switching between making nucleotides by de novo synthesis and scavenging them from the host. This flexibility supports growth and virulence making nucleotide metabolism an attractive therapeutic target.
Riya Joshi   +2 more
wiley   +1 more source

Yersinia pestis Plasminogen Activator

open access: yesBiomolecules, 2020
The Gram-negative bacterium Yersinia pestis causes plague, a fatal flea-borne anthropozoonosis, which can progress to aerosol-transmitted pneumonia. Y. pestis overcomes the innate immunity of its host thanks to many pathogenicity factors, including plasminogen activator, Pla.
Florent Sebbane   +2 more
openaire   +4 more sources

Gasdermin‐Mediated Pyroptosis: Novel Strategies Against Colorectal Cancer

open access: yesCancer Science, Volume 117, Issue 8, Page 2064-2084, August 2026.
Pyroptosis is a double‐edged sword in colorectal cancer (CRC): chronic low‐grade pyroptosis promotes tumor progression, whereas acute induction of pyroptosis in cancer cells triggers antitumor immunity and enhances therapeutic response. Targeting gasdermin‐mediated pyroptosis represents a promising strategy for CRC treatment.
Kaibo Guo   +6 more
wiley   +1 more source

Whole genome alignment file of 368 Yersinia pestis isolates

open access: yes, 2018
Whole genome alignment file of 368 Yersinia pestis ...
chao yang (5581982)
core   +1 more source

Yersinia pestis pathogenicity

open access: yesЖурнал микробиологии, эпидемиологии и иммунобиологии, 2019
Yersinia pestis belongs to those pathogenic bacteria which produce lipopolysaccharide (LPS) having the function of a toxin. In order to make a toxic effect the polymer must be separated from the cell outer membrane and presented to the immunocompetent ...
V. I. Tynyanova   +3 more
doaj   +1 more source

Revisiting Pluripotency Acquisition: A Unified Framework for Reprogramming Strategies

open access: yesDevelopment, Growth &Differentiation, Volume 68, Issue 6, August 2026.
ABSTRACT The traditional hierarchical view of totipotent cells generating diverse lineages of terminally differentiated cells was challenged by the discovery of induced pluripotent stem cells (iPSCs). This breakthrough demonstrated that the ectopic expression of four transcription factors, Oct4, Sox2, Klf4, and c‐Myc (OSKM), can reprogram somatic cells
Anamika Datta, Kunimasa Ohta
wiley   +1 more source

Results of Modeling Experiments in Designing Immuno-Enzyme Test-System for the Detection of Antibodies to Yersinia pestis F1 (ELISA-Ab-F1 Yersinia pestis)

open access: yesПроблемы особо опасных инфекций, 2013
Designed is immuno-enzyme test-system for the detection of antibodies to Yersinia pestis capsular antigen F1 – “ELISA-Ab-F1 Yersinia pestis”.
Z. L. Devdariani   +9 more
doaj   +1 more source

Point of Care Testing and Recombinase Polymerase Amplification

open access: yes
Journal of Clinical Laboratory Analysis, Volume 40, Issue 20, October 2026.
Manfred Weidmann, Ahmed Abd El Wahed
wiley   +1 more source

Vectors and Vector‐Borne Diseases: Biology, Epidemiology and Integrated Control Strategies

open access: yesJournal of Applied Entomology, Volume 150, Issue 7, Page 990-1015, August 2026.
ABSTRACT Vector‐Borne Diseases (VBDs), transmitted by arthropods such as mosquitoes, ticks, fleas and sandflies, represent a significant threat to global health. These diseases can be caused by a variety of pathogens, including bacteria, viruses, protozoa, and helminths.
Roberta Rinaldi   +4 more
wiley   +1 more source

Home - About - Disclaimer - Privacy