Results 131 to 140 of about 624,184 (210)

Subtype-Selective Peptide and Protein Neurotoxic Inhibitors of Nicotinic Acetylcholine Receptors Enhance Proliferation of Patient-Derived Glioblastoma Cell Lines

open access: yesToxins
Glioblastoma multiforme (GBM) is the most aggressive type of brain cancer, with a poor prognosis. GBM cells, which develop in the environment of neural tissue, often exploit neurotransmitters and their receptors to promote their own growth and invasion ...
Elena Gondarenko   +9 more
doaj   +1 more source

Recombinant glycine receptors: stoichiometry and kinetics [PDF]

open access: yes, 2009
Glycine receptors (GlyR) are anion-permeable channels that belong to the pentameric ligand-gated ion channel family. Different GlyR subtypes are known. The main synaptic form is thought to be α1β heteropentamers which mediate fast synaptic inhibition in ...
Krashia, P.
core  

α7 Nicotinic Acetylcholine Receptors May Improve Schwann Cell Regenerating Potential via Metabotropic Signaling Pathways. [PDF]

open access: yesCells, 2023
Botticelli E   +7 more
europepmc   +1 more source

Selective Agonists and Antagonists of α9 Versus α7 Nicotinic Acetylcholine Receptors. [PDF]

open access: yesACS Chem Neurosci, 2022
Papke RL   +4 more
europepmc   +1 more source

Upregtilation of neuronal α7 nicotinic acetylcholine receptors and preconditioning [PDF]

open access: yes, 2007
The upregulation of alpha 7 nicotinic acetylcholine receptors (α7 nAChRs) are putatively reported to play a role in in vivo cortical spreading depression-elicited neuroprotection.
Van Rensburg, Ruan
core  

Effects of ginsenosides, active components of ginseng, on nicotinic acetylcholine receptors expressed in Xenopus oocytes

open access: yes, 2002
We investigated the effects of ginsenosides, the active ingredient of ginseng, on neuronal or muscle-type nicotinic acetylcholine receptor channel activity expressed in Xenopus oocytes after injection of cRNA encoding bovine neuronal α3β4, α7 or human ...
Criado Herrero, Manuel   +9 more
core   +1 more source

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