Results 181 to 190 of about 6,800 (216)
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Screening of Actinobacillus pleuropneumoniae LuxS Inhibitors

Current Microbiology, 2013
LuxS, a conserved bacterial enzyme involved in the activated methyl cycle, catalyzes S-ribosylhomocysteine (SRH) into homocysteine and AI-2 (the inter-species quorum-sensing signal molecule). This enzyme has been reported to be essential for the survival of Actinobacillus pleuropneumoniae in its natural host.
Lu, Li   +6 more
openaire   +2 more sources

Design and analysis of an Actinobacillus pleuropneumoniae transmission experiment

Preventive Veterinary Medicine, 2003
This paper describes a methodology to quantify the transmission of Actinobacillus (A.) pleuropneumoniae from subclinically infected carrier pigs to susceptible contact pigs, and to test the effect of possible interventions on the transmission. The methodology includes the design of a transmission experiment, and a method with which A.
Velthuis, A.G.J.   +4 more
openaire   +4 more sources

Virulence in Actinobacillus pleuropneumoniae and RTX toxins

Trends in Microbiology, 1995
RTX toxins are pore-forming, cytolytic protein toxins that occur widely among pathogenic Gram-negative bacteria. RTX toxins appear to play a direct role in the virulence of Actinobacillus pleuropneumoniae, the etiological agent of porcine pleuropneumonia.
openaire   +3 more sources

Exotoxins of Actinobacillus Pleuropneumoniae

1995
Actinobacillus pleuropneumoniae (previously Haemophilus pleuropneumoniae) is the etiological agent of swine pleuropneumonia, a severe, contagious disease of swine causing important economic losses in industrialized swine production worldwide. The disease is either acute and often fatal or chronic localized and necrotizing.
openaire   +1 more source

Effects of fisetin on virulence of Actinobacillus Pleuropneumoniae

Microbial Pathogenesis
Porcine infectious pleuropneumonia (PCP), caused by Actinobacillus pleuropneumoniae (APP), is a highly infectious respiratory disease of pigs, resulting in significant economic losses to the industry. Apx toxins are cytolytic virulence factors produced by APP.
Qiyun He   +7 more
openaire   +2 more sources

A riboflavin auxotroph of Actinobacillus pleuropneumoniae is attenuated in swine

Infection and Immunity, 1996
Actinobacillus pleuropneumoniae is the etiological agent of a highly contagious and often fatal pleuropneumonia in swine. A riboflavin-requiring mutant of A. pleuropneumoniae serotype 1, designated AP233, was constructed by deleting a portion of the riboflavin biosynthetic operon (ribGBAH) and replacing it with a gene cassette encoding kanamycin ...
T E, Fuller, B J, Thacker, M H, Mulks
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Characterization and immunogenicity of an apxIA mutant of Actinobacillus pleuropneumoniae

Veterinary Microbiology, 2006
Actinobacillus pleuropneumoniae is the aetiological agent of porcine pleuropneumonia, a highly contagious and often fatal disease. A candidate live vaccine strain, potentially capable of cross-serovar protection, was constructed by deleting the section of the apxIA gene coding for the C-terminal segment of ApxI toxin of the A.
Xu, Fuzhou   +8 more
openaire   +4 more sources

Characterization of tetracycline and erythromycin resistance in Actinobacillus pleuropneumoniae

Veterinary Microbiology, 1996
Minimum inhibitory concentrations to tetracycline and erythromycin were determined for nineteen isolates of Actinobacillus pleuropneumoniae of Norwegian origin. The isolates were screened for rRNA methylase determinants (Erm genes) and for the tetracycline resistance Tet B determinant, using oligonucleotide probes, polymerase chain reaction and ...
Y, Wasteson   +3 more
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Immunological properties of Actinobacillus pleuropneumoniae hemolysin I

Veterinary Microbiology, 1991
The 105 kDa hemolysin I protein from Actinobacillus pleuropneumoniae serotype I type strain 4074 (HlyI) was shown by immunoblot analysis to be the predominant immunogenic protein if convalescent field sera or sera from pigs experimentally infected with A. pleuropneumoniae serotype 1 were used.
Frey J, Nicolet J
openaire   +3 more sources

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