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Thrombotic Thrombocytopenic Purpura: Overview on the Last 10 Years of Plasma Exchange Treatment in Hospital São José, Lisbon [PDF]
, 2011 Gutierrez, AS, Tavares, M
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Diagnostic and therapeutic challenges into snakebite-induced thrombotic microangiopathy: a case report and review of the literature. [PDF]
J Med Case RepPanda S, Majhi K, Biswas T, Mishra SB.
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Experience in emergency management of first-episode immune thrombotic thrombocytopenic purpura over the past 21 years: a single-center retrospective study. [PDF]
Front ImmunolZhang L +7 more
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Disintegrin-Like and Metalloprotease With Thrombospondin Type 1 Repeat Motifs 13 (ADAMTS13) Activity in 40 Healthy Dogs. [PDF]
Vet Clin PatholWilkinson AR +2 more
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ADAMTS13 assays and ADAMTS13-deficient mice
Current Opinion in Hematology, 2007Thrombotic thrombocytopenic purpura can be induced by acquired or congenital deficiency of the plasma von Willebrand factor-cleaving protease, ADAMTS13. Measurement of ADAMTS13 activity is important for the diagnosis and treatment of microangiopathies including thrombotic thrombocytopenic purpura.
Toshiyuki, Miyata +4 more
openaire +2 more sources
Blood, 2005
It has now been 3 years since the von Willebrand factor (VWF)–cleaving protease implicated in thrombocytopenic purpura (TTP) pathogenesis was identified as ADAMTS13 (adisintegrin-like and metalloprotease with thrombospondin type 1 motif 13). More than 50 ADAMTS13 mutations resulting in familial TTP have been reported.
Gallia G, Levy +2 more
openaire +2 more sources
It has now been 3 years since the von Willebrand factor (VWF)–cleaving protease implicated in thrombocytopenic purpura (TTP) pathogenesis was identified as ADAMTS13 (adisintegrin-like and metalloprotease with thrombospondin type 1 motif 13). More than 50 ADAMTS13 mutations resulting in familial TTP have been reported.
Gallia G, Levy +2 more
openaire +2 more sources
Current Opinion in Hematology, 2002
Thrombotic thrombocytopenic purpura (TTP) has been a mysterious and deadly disease that often could be treated effectively by plasma exchange, but without real understanding of the underlying pathophysiology. Recent advances now suggest that deficiency of a specific von Willebrand factor (VWF) cleaving protease promotes tissue injury in TTP.
Xinglong, Zheng +2 more
openaire +2 more sources
Thrombotic thrombocytopenic purpura (TTP) has been a mysterious and deadly disease that often could be treated effectively by plasma exchange, but without real understanding of the underlying pathophysiology. Recent advances now suggest that deficiency of a specific von Willebrand factor (VWF) cleaving protease promotes tissue injury in TTP.
Xinglong, Zheng +2 more
openaire +2 more sources
Pharmacogenomics, 2011
The multidomain metalloprotease ADAMTS13 limits thrombus formation via the cleavage of large multimeric forms of von Willebrand factor. Deficiency of functional ADAMTS13 is associated with a number of disease pathologies including thrombotic thrombocytopenic purpura, cardiovascular disease and inflammation.
Sandra Chang, Tseng +1 more
openaire +2 more sources
The multidomain metalloprotease ADAMTS13 limits thrombus formation via the cleavage of large multimeric forms of von Willebrand factor. Deficiency of functional ADAMTS13 is associated with a number of disease pathologies including thrombotic thrombocytopenic purpura, cardiovascular disease and inflammation.
Sandra Chang, Tseng +1 more
openaire +2 more sources