Results 61 to 70 of about 387,299 (303)

Heterogeneity of hepatic stellate cells in a mouse model of non-alcoholic steatohepatitis (NASH)

Hepatology, 2021
In clinical and experimental NASH, the origin of the scar‐forming myofibroblast is the HSC. We used foz/foz mice on a Western diet to characterize in detail the phenotypic changes of HSCs in a NASH model.
S. Rosenthal, Xiao Liu, Souradipta Ganguly, D. Dhar, Martina P. Pasillas, E. Ricciardelli, Rick Z. Li, T. Troutman, T. Kisseleva, C. Glass, D. Brenner   +10 more
semanticscholar   +1 more source

AASLD practice guidance on drug, herbal, and dietary supplement–induced liver injury

, 2022
Hepatology, EarlyView.
Robert J. Fontana, Iris Liou, Adrian Reuben, Ayako Suzuki, M. Isabel Fiel, William Lee, Victor Navarro   +6 more
wiley   +1 more source

Pathohistological characteristics of steatohepatitis on needle biopsy sample from 100 patients [PDF]

Medicinski Podmladak, 2019
Introduction: Steatohepatitis is a form of fatty liver disease, in which there is a deposit of fat and steatosis which occurs in two forms: alcoholic and non-alcoholic fatty liver disease.
Milićević Anđela, Lazović Nevena, Tomanović Nada   +2 more
doaj  

Effect of quercetin on the clinical course of non-alcoholic steatohepatitis and diabetic kidney disease against comprehensive treatment of type 2 diabetes patients

Mìžnarodnij Endokrinologìčnij Žurnal, 2020
Background. The purpose of the study was to determine the probable effect of a complex of metformin, rosuvastatin, essential phospholipids, and quercetin on the clinical course of non-alcoholic steatohepatitis, diabetic kidney disease, type 2 diabetes ...
O.S. Khukhlina, A.A. Antoniv, Z.Ya. Kotsiubiichuk   +2 more
doaj   +1 more source

Gene expression profiling unravels cancer-related hepatic molecular signatures in steatohepatitis but not in steatosis. [PDF]

PLoS ONE, 2012
BACKGROUND: Pathogenesis and factors for determining progression of alcoholic and non-alcoholic steatosis to steatohepatitis with risk of further progression to liver cirrhosis and cancer are poorly understood.
Julia Starmann, Maria Fälth, Walter Spindelböck, Katja-Lauren Lanz, Carolin Lackner, Kurt Zatloukal, Michael Trauner, Holger Sültmann   +7 more
doaj   +1 more source

Letter to the Editor: LO2, a misidentified cell line: Some data should be interpreted with caution

, 2022
Hepatology, EarlyView.
Ralf Weiskirchen
wiley   +1 more source

Alcoholic liver disease and alcoholism: two diseases, one problem

Медицинский совет, 2013
The article highlights alcoholic liver disease manifested in steatosis/steatohepatitis/fibrosis, which further transforms into cirrhosis of the liver.
A. A. Samsonov, E. Y. Plotnikova, I. N. Nikushkina, E. A. Talitskaya, M. V. Krasnova, O. A. Krasnov   +5 more
doaj   +1 more source

Monozygotic twins with NASH cirrhosis: cumulative effect of multiple single nucleotide polymorphisms?

Annals of Hepatology, 2016
Multiple genetic and environmental factors interact to determine an individual’s predisposition to non-alcoholic fatty liver disease and its phenotypic characteristics. Association studies have found a number of alleles associated with the development of
Jane I. Grove, Mark Austin, Jeremy Tibble, Guruprasad P. Aithal, Sumita Verma   +4 more
doaj   +1 more source

Clinical impact of sexual dimorphism in non‐alcoholic fatty liver disease (NAFLD) and non‐alcoholic steatohepatitis (NASH)

Liver international (Print), 2021
NAFLD/NASH is a sex‐dimorphic disease, with a general higher prevalence in men. Women are at reduced risk of NAFLD compared to men in fertile age, whereas after menopause women have a comparable prevalence of NAFLD as men.
P. Burra, D. Bizzaro, A. Gonţa, S. Shalaby, M. Gambato, M. Morelli, S. Trapani, A. Floreani, F. Marra, M. Brunetto, G. Taliani, E. Villa   +11 more
semanticscholar   +1 more source

The therapeutic landscape of non-alcoholic steatohepatitis. [PDF]

, 2016
Non-alcoholic steatohepatitis (NASH) is characterized by lobular inflammation and hepatocellular ballooning, and may be associated with liver fibrosis leading to cirrhosis and its complications.
Perazzo, Hugo, Hugo Perazzo, Dufour, Jean-François, Dufour, J. F., Jean‐François Dufour   +4 more
core   +1 more source