Results 81 to 90 of about 358 (125)

High-Expression of AMAP1 and EPB41L5 Proteins Correlates with Lymph Node Metastasis and Survival Rate after Surgical Therapy of HNSCC. [an abstract of entire text]

open access: yesHigh-Expression of AMAP1 and EPB41L5 Proteins Correlates with Lymph Node Metastasis and Survival Rate after Surgical Therapy of HNSCC. [an abstract of entire text]
(主査) 教授 佐邊 壽孝, 教授 野口 昌幸, 准教授 濱田 淳一, 教授 福田 諭 医学研究科(医学専攻) この博士論文全文の閲覧方法については、以下のサイトをご参照ください。 配架番号:2093 https://www.lib.hokudai.ac.jp/dissertations/copy-guides/
openaire   +1 more source

Peptides as integrative modulators for clinical prognosis and targeted therapy in pancreatic cancer. [PDF]

open access: yesDiscov Oncol
Yadav B   +7 more
europepmc   +1 more source

Abstract 2788 Chemokine Switching via ARF6-AMAP1: A Key Mechanism of Immune Evasion in Pancreatic Cancer

open access: yesJournal of Biological Chemistry
Ari Hashimoto   +8 more
openaire   +1 more source

High-Expression of AMAP1 and EPB41L5 Proteins Correlates with Lymph Node Metastasis and Survival Rate after Surgical Therapy of HNSCC. [an abstract of dissertation and a summary of dissertation review]

open access: yesHigh-Expression of AMAP1 and EPB41L5 Proteins Correlates with Lymph Node Metastasis and Survival Rate after Surgical Therapy of HNSCC. [an abstract of dissertation and a summary of dissertation review]
(主査) 教授 佐邊 壽孝, 教授 野口 昌幸, 准教授 濱田 淳一, 教授 福田 諭 医学研究科(医学専攻) 配架番号 ...
openaire   +1 more source

Polyphenols in Pancreatic Cancer Management: Exploring the Roles and Mechanisms of Resveratrol and Epigallocatechin. [PDF]

open access: yesOncol Res
de la Garza-Kalife DA   +7 more
europepmc   +1 more source

Frequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model

open access: yesFrequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model
Background: The small GTPase Arf6 and its downstream effector AMAP1 (also called ASAP1/DDEF1) constitute a signaling pathway promoting cell invasion, in which AMAP1 interacts with several different proteins, including PRKD2, EPB41L5, paxillin, and cortactin.
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Epithelial-specific histone modification of the miR-96/182 locus targeting AMAP1 mRNA predisposes p53 to suppress cell invasion in epithelial cells

open access: yesEpithelial-specific histone modification of the miR-96/182 locus targeting AMAP1 mRNA predisposes p53 to suppress cell invasion in epithelial cells
Background: TP53 mutations in cancer cells often evoke cell invasiveness, whereas fibroblasts show invasiveness in the presence of intact TP53. AMAP1 (also called DDEF1 or ASAP1) is a downstream effector of ARF6 and is essential for the ARF6-driven cell-invasive phenotype.
openaire   +1 more source

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