Results 81 to 90 of about 358 (125)
ADP-ribosylation factor 6 expression increase in oesophageal adenocarcinoma suggests a potential biomarker role for it. [PDF]
Kanamarlapudi V +2 more
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(主査) 教授 佐邊 壽孝, 教授 野口 昌幸, 准教授 濱田 淳一, 教授 福田 諭 医学研究科(医学専攻) この博士論文全文の閲覧方法については、以下のサイトをご参照ください。 配架番号:2093 https://www.lib.hokudai.ac.jp/dissertations/copy-guides/
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Mechanisms of Immune Cell Dysregulation in Pancreatic Ductal Adenocarcinoma. [PDF]
Ahmady-Nield F, Luwor RB, Kannourakis G.
europepmc +1 more source
Peptides as integrative modulators for clinical prognosis and targeted therapy in pancreatic cancer. [PDF]
Yadav B +7 more
europepmc +1 more source
(主査) 教授 佐邊 壽孝, 教授 野口 昌幸, 准教授 濱田 淳一, 教授 福田 諭 医学研究科(医学専攻) 配架番号 ...
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Polyphenols in Pancreatic Cancer Management: Exploring the Roles and Mechanisms of Resveratrol and Epigallocatechin. [PDF]
de la Garza-Kalife DA +7 more
europepmc +1 more source
Identification of Short Amino Acid Sequences That Correlate with Cytoplasmic Retention of Human Proteins. [PDF]
Brown JC, Wang B.
europepmc +1 more source
Background: The small GTPase Arf6 and its downstream effector AMAP1 (also called ASAP1/DDEF1) constitute a signaling pathway promoting cell invasion, in which AMAP1 interacts with several different proteins, including PRKD2, EPB41L5, paxillin, and cortactin.
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Background: TP53 mutations in cancer cells often evoke cell invasiveness, whereas fibroblasts show invasiveness in the presence of intact TP53. AMAP1 (also called DDEF1 or ASAP1) is a downstream effector of ARF6 and is essential for the ARF6-driven cell-invasive phenotype.
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