Results 181 to 190 of about 2,474,551 (357)

Discovery of ultrafast myosin, its amino acid sequence, and structural features. [PDF]

open access: yesProc Natl Acad Sci U S A, 2022
Haraguchi T   +9 more
europepmc   +1 more source

Neuropeptide Y: complete amino acid sequence of the brain peptide.

open access: yesProceedings of the National Academy of Sciences of the United States of America, 1982
K. Tatemoto
semanticscholar   +1 more source

Evaluating the involvement of autolysosomes in the nuclear translocation of fluorescent proteins

open access: yesFEBS Open Bio, EarlyView.
Endogenously expressed fluorescent proteins can be degraded by autophagy and transported to cell nuclei via the nuclear pore complex. But in some cell lines, for example, HeLa cells which are positive for immunoreactivity of a receptor ligand, such as UCN I, in cell nuclei, fusion of autolysosome with the nuclear envelope is involved in the nuclear ...
Keiichi Ikeda
wiley   +1 more source

Aging Is a Key Driver for Adult Acute Myeloid Leukemia

open access: yesAging and Cancer, EarlyView.
Acute myeloid leukemia (AML) is a classical age‐related hematologic malignancy, and a key driver of AML is aging, which profoundly regulates intrinsic factors such as genomic instability, epigenetic reprogramming, and metabolic dysregulation, and alters bone marrow microenvironment.
Rong Yin, Haojian Zhang
wiley   +1 more source

Targeted modulation of protein liquid-liquid phase separation by evolution of amino-acid sequence. [PDF]

open access: yesPLoS Comput Biol, 2021
Lichtinger SM   +3 more
europepmc   +1 more source

A method for the determination of amino acid sequence in peptides.

open access: yesArchives of biochemistry, 1949
P. Edman   +3 more
semanticscholar   +1 more source

Mutant NPM1 in Acute Myeloid Leukemia Initiation and Maintenance

open access: yesAging and Cancer, EarlyView.
NPM1 mutations drive acute myeloid leukemia by acting as neomorphic transcriptional regulators that cooperate with Menin–MLL and XPO1 to sustain HOX/MEIS1 expression and block differentiation. Targeting these mutant‐specific transcriptional dependencies provides a rational therapeutic strategy for NPM1‐mutated AML.
Yanan Jiang   +3 more
wiley   +1 more source

Taxonomy of Mitochondrial Cytochrome B Proteins of the Same Amino Acid Sequence Length. [PDF]

open access: yesScientificWorldJournal, 2021
Zamyatnin AA   +2 more
europepmc   +1 more source

Super‐Refractory Status Epilepticus (SRSE) in a Patient With Compound Heterozygous OPA1 Variants: Case Report and Literature Review

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective Super‐Refractory Status Epilepticus (SRSE) is a rare, life‐threatening neurological emergency with unclear etiology in many cases. Mitochondrial dysfunction, often due to disease‐causing genetic variants, is increasingly recognized as a cause, with each gene producing distinct pathophysiological mechanisms.
Pouria Mohammadi   +2 more
wiley   +1 more source

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