Results 211 to 220 of about 99,629 (284)

β‐Adrenergic Receptor Activation Modulates the Induction of Complex Spike‐Dependent LTP by Regulating Multiple Forms of Heterosynaptic Plasticity

open access: yesHippocampus, Volume 35, Issue 6, November 2025.
ABSTRACT Norepinephrine, acting through β‐adrenergic receptors (β‐ARs), has a key role in hippocampus‐dependent forms of learning. Although β‐AR activation also facilitates the induction of Hebbian LTP, recent findings indicate that a non‐Hebbian form of synaptic plasticity, known as behavioral timescale synaptic plasticity (BTSP), underlies ...
Thomas J. O'Dell
wiley   +1 more source

Erratum: Sustained postsynaptic kainate receptor activation downregulates AMPA receptor surface expression and induces hippocampal LTD. [PDF]

open access: yesiScience
Nair JD   +8 more
europepmc   +1 more source

Notes on the History of In Vitro Hippocampal Electrophysiology and LTP: Personal Reflections

open access: yesHippocampus, Volume 35, Issue 6, November 2025.
ABSTRACT This essay describes the development of the in vitro hippocampal slice technique and my small contributions to it and to influencing Roger Nicoll's early interests in the hippocampus and LTP. My Ph.D. work at Harvard with Timothy Teyler was on field potential studies of synaptic plasticity, including LTP, in the rat in vitro hippocampal slice.
Bradley E. Alger
wiley   +1 more source

The Lurcher Mutation Identifies δ2 as an AMPA/Kainate Receptor-Like Channel That Is Potentiated by Ca2+ [PDF]

open access: bronze, 2000
Lonnie P. Wollmuth   +5 more
openalex   +1 more source

Use of proteoliposomes to generate phage antibodies against native AMPA receptor [PDF]

open access: bronze, 2000
Lene K. Jespersen   +4 more
openalex   +1 more source

Clinical and Neurodevelopmental Characteristics of Paralogous Gain‐of‐Function Variants at GRIA2 p.Gly792 and GRIA3 p.Gly803

open access: yesClinical Genetics, Volume 108, Issue 5, Page 553-565, November 2025.
Key features of paralogous GRIA2 and GRIA3 gain‐of‐function variants. ABSTRACT GRIA‐related disorders arise from disease‐causing variants in GRIA1, GRIA2, GRIA3, or GRIA4 that encode α‐amino‐3‐hydroxy‐5‐methyl‐4‐isoxazole propionic acid (AMPA)‐type glutamate receptors (AMPARs).
Emilie Sjøstrøm   +24 more
wiley   +1 more source

PDZ Proteins Interacting with C-Terminal GluR2/3 Are Involved in a PKC-Dependent Regulation of AMPA Receptors at Hippocampal Synapses

open access: bronze, 2000
Michael I. Daw   +7 more
openalex   +1 more source

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