Results 201 to 210 of about 8,073,410 (232)
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Biomarkers in ANCA-Associated Vasculitis
Current Rheumatology Reports, 2013Despite recent advances in the treatment of antineutrophil cytoplasmic autoantibody (ANCA)-associated vasculitis (AAV), relapse remains common and patients often experience a variable clinical course after initial treatment. New biomarkers are needed to aid the management of these complex diseases.
Lindsay, Lally, Robert F, Spiera
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Giornale italiano di nefrologia : organo ufficiale della Societa italiana di nefrologia, 2002
ANCA-associated vasculitis. The term "antineutrophil cytoplasm antibody (ANCA)- associated vasculitis" (AASV) ihighers generally used to include primary vasculitis syndromes in which circulating ANCA against proteinase 3 (PR3) and myeloperoxidase (MPO) are commonly found.
SINICO, RENATO ALBERTO +3 more
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ANCA-associated vasculitis. The term "antineutrophil cytoplasm antibody (ANCA)- associated vasculitis" (AASV) ihighers generally used to include primary vasculitis syndromes in which circulating ANCA against proteinase 3 (PR3) and myeloperoxidase (MPO) are commonly found.
SINICO, RENATO ALBERTO +3 more
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Treatment of ANCA-associated vasculitis
Nature Reviews Nephrology, 2013Antineutrophil cytoplasmic autoantibody (ANCA)-associated diseases are small-vessel vasculitides, encompassing granulomatosis with polyangiitis (formerly Wegener's granulomatosis), microscopic polyangiitis and eosinophilic granulomatosis with polyangiitis.
Ulf, Schönermarck +2 more
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Epidemiology of ANCA-associated Vasculitis
Rheumatic Disease Clinics of North America, 2010The epidemiology of the antineutrophil cytoplasm antibody (ANCA)-associated vasculitides (AAV), comprising Wegener's granulomatosis, microscopic polyangiitis, and Churg-Strauss syndrome, poses considerable challenges to epidemiologists. These challenges include the difficulty of defining a case with a lack of clear distinction between the different ...
Ntatsaki, Eleana +2 more
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Treatment of ANCA-Associated Vasculitis
New England Journal of Medicine, 2003Vasculitis is defined by inflammation of the blood-vessel wall and forms the pathological foundation of a diverse group of individual disease entities. Among the primary systemic vasculitic diseases, Wegener's granulomatosis and microscopic polyangiitis share several common features, including pulmonary capillaritis, pauci-immune focal crescentic ...
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Vasculitis for the internist: focus on ANCA-associated vasculitis
Internal and Emergency Medicine, 2017Vasculitides comprise several diseases affecting vessels of different sizes, mainly arteries, but also capillaries and veins. Antineutrophil cytoplasm antibody-associated vasculitides (AAVs) belong to the systemic necrotizing small-vessel vasculitis group that comprises granulomatosis with polyangiitis, microscopic polyangiitis and eosinophilic ...
Benjamin, Chaigne, Loïc, Guillevin
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The History of ANCA-associated Vasculitis
Rheumatic Disease Clinics of North America, 2010An essential early step toward understanding vasculitis was recognition in 1948 of the differences between the small artery disease of polyarteritis, essentially sparing the glomerulus and lungs, and disease of glomerular vessels and small veins, often involving the lungs.
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Autoantibodies in ANCA-associated Vasculitis
Rheumatic Disease Clinics of North America, 2010Antineutrophil cytoplasmic autoantibody (ANCA)-associated vasculitides are systemic or more limited conditions characterized by necrotizing destruction of small and medium-sized vessels (eg, capillaries, venules, and arterioles). ANCAs are the most predominant autoantibodies in patients affected by vasculitis, but other autoantibodies may also occur ...
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ANCA associated pulmonary fibrosis: vasculitis or not vasculitis
Current Opinion in Pulmonary MedicinePurpose of review The intersection of antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) and interstitial lung disease (ILD) represents a complex and increasingly recognized clinical challenge. This review aims to summarize current understanding, highlight diagnostic and therapeutic approaches, and identify ...
Codes-Méndez, H +4 more
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