Results 181 to 190 of about 4,449,758 (301)

Adaptor protein CIN85 potentiates the motility of osteosarcoma cells via the Akt/mTOR and MMP2‐COL3A1 axis

open access: yesMolecular Oncology, EarlyView.
CIN85 is highly expressed in osteosarcoma, particularly in metastatic lesions. Its overexpression increases cell migration and Matrigel invasion, while silencing CIN85 suppresses these behaviors. Transcriptome analysis shows that CIN85 regulates MMP2, COL3A1, and Akt/mTOR signaling. Targeting these pathways reverses CIN85‐induced motility, highlighting
Iryna Horak   +10 more
wiley   +1 more source

Open Access Journals in Library and Information Science: The Story so Far

open access: yes, 2012
Purpose: Internet has triggered the growth of scholarly publications and every discipline is witnessing an unremitting growth in the scholarly market. Open access, the product of Internet has also captured the global disciplines.
Rufai, Reyaz; University of Kashmir   +2 more
core  

Longitudinal circulating tumor DNA profiling in patients with advanced endometrial cancer using an off‐the‐shelf targeted NGS panel

open access: yesMolecular Oncology, EarlyView.
Intratumour heterogeneity complicates precision management of advanced endometrial cancer. Circulating tumor DNA (ctDNA) offers a minimally invasive strategy to capture tumor evolution and therapeutic resistance. Here, we compare tumor‐agnostic NGS with tumor‐informed ddPCR, outlining their relative sensitivity, concordance, and clinical implications ...
Carlos Casas‐Arozamena   +15 more
wiley   +1 more source

The Islamic World Science Citation Center: A New Scientometrics System for Evaluating Research Performance in OIC Region

open access: yes, 2012
This article deals with the establishment of the Islamic World Science Citation Center (ISC) as the first citation system in the Islamic countries. It attempts to describe the mapping of ISC the way it has been established.
Mehrad, J.; Ph.D., Regional Information Center for Science and Technology   +1 more
core  

Interaction of HS1BP3 with cortactin modulates TKS5 localisation, cell secretion and cancer malignancy

open access: yesMolecular Oncology, EarlyView.
Here, we demonstrate that HS1BP3 interacts with Cortactin through a proline‐rich region (PRR3.1) and show that this interaction, and HS1BP3 itself, promote cancer cell proliferation and invasion. Inhibition of this interaction leads to build‐up of TKS5 in multivesicular endosomes and altered secretion of CD63 and CD9, providing an explanation for the ...
Arja Arnesen Løchen   +9 more
wiley   +1 more source

JISTaP Vol.1 No.3

open access: yes, 2018
Korea Institute of Science and Technology Information
core  

Home - About - Disclaimer - Privacy