Results 261 to 270 of about 5,964,638 (304)
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The Angiotensin II Receptor and the Actions of Angiotensin II
Journal of Cardiovascular Pharmacology, 1990Angiotensin II (Ang II) is a potent effector peptide of the renin-angiotensin system that exerts a wide variety of physiological actions on the cardiovascular, renal, endocrine, and central and peripheral nervous systems. Angiotensin exerts its actions by binding to specific receptors in the plasma membrane of various tissues.
M J, Peach, D E, Dostal
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Human Angiotensin II Receptors are Regulated By Angiotensin II
Clinical and Experimental Hypertension. Part A: Theory and Practice, 1988Specific angiotensin II (ANG II) binding on human platelets in man was measured to examine ANG II receptor regulation in man. Eight normal volunteers received either a high, or a low sodium diet, or a low sodium diet together with the ANG I converting enzyme inhibitor enalapril.
J F, Mann, M, Leidig, E, Ritz
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Conversion of Angiotensin I to Angiotensin II
Nature, 1967Results obtained with the blood bathed organ technique indicate that angiotensin I is converted rapidly to angiotensin II in the pulmonary circulation and not by an enzyme in the blood.
K K, Ng, J R, Vane
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Inflammation and angiotensin II
The International Journal of Biochemistry & Cell Biology, 2003Angiotensin II (AngII), the major effector peptide of renin-angiotensin system (RAS), is now recognized as a growth factor that regulates cell growth and fibrosis, besides being a physiological mediator restoring circulatory integrity. In the last few years, a large number of experimental studies has further demonstrated that AngII is involved in key ...
Yusuke, Suzuki +5 more
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Angiotensin II and atherosclerosis
The American Journal of Cardiology, 2001Numerous clinical and laboratory data are now available supporting the hypothesis that the renin-angiotensin system is mechanistically relevant in the pathogenesis of atherosclerosis. The traditional role of the renin-angiotensin system in the context of blood pressure regulation has been modified to incorporate the concept that angiotensin II (Ang II)
D, Weiss, D, Sorescu, W R, Taylor
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Conversion of angiotensin I to angiotensin II
The American Journal of Medicine, 1976The angiotensin I converting enzyme has two important functions: it inactivates bradykinin and converts angiotensin I to angiotensin II. Inhibition of the enzyme blocks the renin-angiotensin system and decreases systemic blood pressure if the pressure is maintained or increased by renin. The enzyme occurs in a variety of tissues and cell forms.
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CONVERSION OF ANGIOTENSIN I TO ANGIOTENSIN II IN SHEEP
Clinical and Experimental Pharmacology and Physiology, 1978SUMMARY1. The methodology for measurement of angiotensin I in whole blood is described.2. Angiotensin I was measured in arterial and venous blood samples from sodium‐loaded, sodium‐replete and sodium‐depleted sheep. Venous blood concentrations were higher than arterial angiotensin I concentrations.
R T, Fernley +5 more
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Is the adrenal angiotensin receptor angiotensin II — or angiotensin III like?
Acta Endocrinologica, 1983Abstract. In order to determine whether the adrenal receptor is primarily directed at angiotensin II (All) or angiotensin III (AIII) the following in vitro experiments were performed examining aldosterone responsiveness in isolated glomerulosa cells.
L M, Braley +3 more
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Angiotensin II and the adrenal.
Clinical and experimental pharmacology & physiology, 19961. Angiotensin II (AngII) evokes a variety of physiological responses in the adrenal gland. It is the major regulator of aldosterone secretion, in the medulla it enhances catecholamine release and it exerts trophic effects in the adrenal and stimulates growth factor secretion. 2.
G. Giacchetti +4 more
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Journal of Cardiovascular Pharmacology, 1991
The consequence of a sustained rise in blood pressure is an adaptive change in the structure of the heart and the vasculature. In humans, the left ventricle, aorta, and medium-sized arteries undergo hypertrophy whereas the changes in resistance vessels are unclear at present.
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The consequence of a sustained rise in blood pressure is an adaptive change in the structure of the heart and the vasculature. In humans, the left ventricle, aorta, and medium-sized arteries undergo hypertrophy whereas the changes in resistance vessels are unclear at present.
openaire +2 more sources