Results 281 to 290 of about 125,197 (343)

Locked nucleic acid-modified antisense oligonucleotides attenuate scar hyperplasia through targeted inhibition of CTGF. [PDF]

Front Pharmacol
Li J, Wu X, Yang Y, Mao R, Li Z, Zhang X, Wei W, Wang W, Li H, Zhou H, Yang C.   +10 more
europepmc   +1 more source

<i>SNORA13</i> antisense oligonucleotides enhances the therapeutical effects of 5-fluorouracil in colon adenocarcinoma. [PDF]

Front Pharmacol
Wang Y, An H, Zhang Y, Lyu QR, Zhang Z.
europepmc   +1 more source

Harnessing BET-Bromodomain Assisted Nuclear Import for Targeted Subcellular Localization and Enhanced Efficacy of Antisense Oligonucleotides. [PDF]

J Am Chem Soc
Kashyap D, Cadeddu M, Oliver PL, Milne TA, Booth MJ.   +4 more
europepmc   +1 more source

Evolution of antisense oligonucleotides: navigating nucleic acid chemistry and delivery challenges. [PDF]

Expert Opin Drug Discov
Ruchi R, Raman GM, Kumar V, Bahal R.
europepmc   +1 more source

Topological Switch-OFF and β-Galactosidase-Triggered Switch-ON of Cyclic Antisense Oligonucleotides via CuAAC for Controlled RNA Cleavage. [PDF]

Bioconjug Chem
Miyaji K, Takeuchi K, Seio K.
europepmc   +1 more source

Glycogen for Smyd3-antisense oligonucleotide delivery and enhanced liver cancer gene therapy

Ning Bai, Yi Huang, Jie Shen, Yefan Peng, Huijie Zhang, Deqiang Hou   +5 more
openalex   +2 more sources

Anti sense and sensibility : renal and skin effects of (antisense) oligonucleotides

, 2017
Laurens van der Meer, van
openalex   +1 more source

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Antisense Oligonucleotides

Nephron Experimental Nephrology, 1998
Antisense technology was developed to inhibit gene expression by utilizing an oligonucleotide complementary to the mRNA which encodes the target gene. There are a few possible mechanisms for the inhibitory effects of antisense oligonucleotides. Among them, degradation of mRNA by RNase H is considered to be the major mechanism of action for antisense ...
N, Kashihara, Y, Maeshima, H, Makino
openaire   +2 more sources

Antisense-Oligonucleotide Therapy

New England Journal of Medicine, 1996
Many pharmacologic advances involve creating compounds that bind and disable proteins. Such compounds include propranolol, which blocks the β-adrenergic receptor; cimetidine, which blocks the H2 receptor; calcium-channel blockers; angiotensin-converting–enzyme inhibitors; and inhibitors of the H+/K+–ATPase pump.
F K, Askari, W M, McDonnell
openaire   +2 more sources