Results 81 to 90 of about 2,626,293 (190)

Atrial tachycardia in a patient with arrhythmogenic right ventricular cardiomyopathy/dysplasia

open access: yesJournal of Arrhythmia, 2013
We describe a 49-year-old woman with atrial tachycardia (AT) and arrhythmogenic right ventricular cardiomyopathy/dysplasia. Cardiac magnetic resonance images showed a markedly dilated right atrium and right ventricle.
Hiroaki Mano   +11 more
doaj   +1 more source

Intercalated disk structure, tissue heterogeneity and ion channel distribution modulate conduction and local calcium influx

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend A, three‐dimensional representation of cardiac intercalated disk, reduced to a 100‐partition, computationally efficient network representation. B, ion dynamics within intercalated disk extracellular cleft space. C, rapid pacing leads to localized conduction block in tissue with regions of perturbed intercalated disk (ID ...
Nicolae Moise   +6 more
wiley   +1 more source

Ricerca di mutazioni nel gene CTNNA3 candidato per la cardiomiopatia aritmogena del ventricolo destro [PDF]

open access: yes, 2008
Risultati di un tirocinio dove è stato effettuato uno screening di mutazioni in un gene candidato in 10 pazienti.
Martinati, Francesco
core  

Genetics of arrhythmogenic right ventricular cardiomyopathy

open access: yes, 2013
Arrhythmogenic right ventricular cardiomyopathy is a rare clinical entity characterised by fibro-fatty replacement of myocardium, mainly involving right ventricular free wall, leading to malignant electrical instability and sudden cardiac death.
Partemi, Sara   +2 more
core   +1 more source

The multicellular environment of the re‐entrant circuit in post‐myocardial infarction ventricular tachycardia: Beyond fibrosis

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Schematic overview of the temporal evolution of the post‐myocardial infarction substrate leading to ventricular tachycardia. Acute ischaemic injury initiates metabolic and electrophysiological disruption, followed by progressive inflammatory, fibrotic and structural remodelling within the infarct border zone.
Mohammed Obeidat   +6 more
wiley   +1 more source

Arrhythmogenic ventricular cardiomyopathy and sudden cardiac death: Left or right? [PDF]

open access: yes, 2017
Arrhythmogenic right ventricular cardiomyopathy is a leading cause of sudden cardiac death among athletes in Italy and the Mediterranean region. Although it often involves the right ventricle causing scarring, dilation, systolic impairment with aneurysm ...
AlJaroudi, Wael A.   +4 more
core   +1 more source

Translating cardiovascular ion channel and Ca2+ signalling mechanisms into therapeutic insights

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend This white paper integrates mechanistic discoveries across ion channel biology, Ca2+ signalling and multiscale cardiovascular physiology to highlight new opportunities for accelerating research and guiding next‐generation therapies. Printed with permission from ®Anita Impagliazzo Medical Illustration. [Correction added on 2 March
Silvia Marchianò   +18 more
wiley   +1 more source

Arrhythmogenic Right Ventricular Cardiomyopathy Caused by a Novel Frameshift Mutation

open access: yes, 2016
Arrhythmogenic right ventricular cardiomyopathy is a rare cardiomyopathy that might be asymptomatic or symptomatic, causing palpations or syncope, and might lead to sudden cardiac death. It is recommended that physical exertion be reduced.
Refaat, Marwan M.   +5 more
core   +1 more source

Arrhythmogenic Right Ventricular Cardiomyopathy

open access: yes, 2022
Arrhythmogenic right ventricular cardiomyopathy (ARVC) encompasses a group of conditions characterized by right ventricular fibrofatty infiltration, with a predominant arrhythmic presentation.
Calkins, Hugh   +6 more
core   +1 more source

Advanced imaging and multi‐omics to characterize cardiomyocyte ageing and its electrocardiographic imprint in PKP2 arrhythmogenic cardiomyopathy

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend We used a murine model of cardiomyocyte‐specific conditional knockout of plakophilin 2 (PKP2cKO, middle panel). Using advanced imaging technology, we unveiled distinct hallmarks of premature ageing in PKP2cKO cardiomyocytes, including DNA damage and loss of repressed heterochromatin at the nuclear periphery, and mitochondrial ...
Giorgia Bertoli   +12 more
wiley   +1 more source

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