Results 131 to 140 of about 6,998 (166)

Treatment of atypical hemolytic uremic syndrome with eculizumab in a patient presenting with neuropsychiatric prodrome: a case report. [PDF]

Front Immunol
Yang H, Zhang J, Wu H, Xu H, Xu Z.
europepmc   +1 more source

Clinical and genetic characteristics of patients diagnosed with atypical hemolytic uremic syndrome (aHUS): epidemiological data from the Belgian cohort of the Global aHUS Registry. [PDF]

J Nephrol
Massart A, Weekers L, Claes KJ, Van Meerhaeghe T, Snauwaert E, Mekahli D, Goffin E, Collard L, Godefroid N, Adams B, Van Cauwelaert S, Van Hoeck K, Block S, Al-Dakkak I, Dahan K, Stordeur P, Vande Walle J.   +16 more
europepmc   +1 more source

Real-World Effectiveness of Ravulizumab Among C5 Inhibitor-Naive Patients With Atypical Hemolytic Uremic Syndrome: A Physician Panel-Based Chart Review (aHUS IMPACT Study). [PDF]

Kidney Med
Hanna RM, Chaturvedi S, Ong ML, Nag A, Song R, Huynh L, Burdeau JA, Duh MS, Wang Y.   +8 more
europepmc   +1 more source

Effectiveness and Safety of Switching to Ravulizumab From Eculizumab in Kidney Transplant Recipients With Atypical Hemolytic Uremic Syndrome: A Global aHUS Registry Analysis. [PDF]

Clin Transplant
Gaeckler A, Al-Dakkak I, Saval N, Dieperink HH, Eygenraam M, Greenbaum LA, Isbel N, Walle JV.   +7 more
europepmc   +1 more source

Transplantation in Atypical Hemolytic Uremic Syndrome [PDF]

Seminars in Thrombosis and Hemostasis, 2010
Atypical hemolytic uremic syndrome (aHUS) is a disease characterized by overactivation of complement. Recurrence following renal transplantation is determined by a genetic predisposition. Genetic screening of all individuals with aHUS should be performed prior to listing for transplantation. Individuals with isolated mutations in MCP have a low risk of
Hannu Jalanko, David Kavanagh, Tim Goodship   +2 more
exaly   +4 more sources

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Plasmatherapy in Atypical Hemolytic Uremic Syndrome

Seminars in Thrombosis and Hemostasis, 2010
Plasmatherapy has become empirically first-line treatment in atypical hemolytic uremic syndrome (aHUS), although no prospective controlled trials have been conducted. Patients with mutations that induce complete or partial factor H (FH) quantitative deficiency may be controlled by plasma infusions (PI), but plasma exchanges appear more efficient than ...
Anne-Laure Sellier-Leclerc, Chantal Loirat   +1 more
exaly   +3 more sources

Atypical Hemolytic–Uremic Syndrome

New England Journal of Medicine, 2009
The hemolytic–uremic syndrome, which is characterized by nonimmune hemolytic anemia, thrombocytopenia, and renal impairment, occurs most frequently in young children. Most cases are secondary to infection with Escherichia coli O157:H7 and other Shiga-toxin–producing strains.
Marina, Noris, Giuseppe, Remuzzi
openaire   +2 more sources

Pathogenesis of Atypical Hemolytic Uremic Syndrome

Journal of Atherosclerosis and Thrombosis, 2019
Atypical hemolytic uremic syndrome (aHUS) is a type of thrombotic microangiopathy (TMA) defined by thrombocytopenia, microangiopathic hemolytic anemia, and renal failure. aHUS is caused by uncontrolled complement activation in the alternative pathway (AP).
Masaomi Nangaku, Yoichirô Ikeda
exaly   +4 more sources

Atypical hemolytic uremic syndrome

Current Opinion in Hematology, 2010
The last few years revealed a molecular distinction between thrombotic thrombocytopenic purpura, a disease characterized by a lack of ADAMTS13 activity, and atypical hemolytic uremic syndrome (aHUS), a disease of complement overactivation. Many different predisposing genetic factors resulting in complement overactivation have been described in aHUS ...
Kavanagh D, Goodship THJ
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Atypical hemolytic uremic syndrome

Pediatrics International, 1995
AbstractThe pathogenesis of atypical uremic syndrome (HUS), which is rarely encountered in childhood, is poorly understood and its mortality and morbidity rates are high. A wide variety of therapeutic approaches has been attempted and the literature contains numerous conflicting reports about the results of these approaches.
KAVUKÇU S., İRKEN G., OLGUN N., GÜÇLÜ A.   +3 more
openaire   +4 more sources