Results 91 to 100 of about 333,490 (283)

Manual for the Fish Population Surveys (DOC9 Package) for the District Fisheries Analysis System (FAS) [PDF]

open access: yes, 1990
Update of Aquatic Biology Technical Report 87/11; final report of project F-69-R (1-3), Data Base Management and Analysis of Fisheries in ImpoundmentsReport issued on: issued October 1990INHS Technical Report prepared for Illinois Department ...
Austen, Douglas J.   +2 more
core  

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

RSS Tracking Control for AVs Under Bayesian-Network-Based Intelligent Learning Scheme

open access: yesActuators
In complex real-world traffic environments, the task of automatic lane changing becomes extremely challenging for vehicle control systems. Traditional control methods often lack the flexibility and intelligence to accurately capture and respond to ...
Kun Zhang, Kezhen Han, Nanbin Zhao
doaj   +1 more source

Emerging role of ARHGAP29 in melanoma cell phenotype switching

open access: yesMolecular Oncology, EarlyView.
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster   +3 more
wiley   +1 more source

In vitro properties of patient serum predict clinical outcome after high dose rate brachytherapy of hepatocellular carcinoma

open access: yesMolecular Oncology, EarlyView.
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser   +14 more
wiley   +1 more source

Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine

open access: yesMolecular Oncology, EarlyView.
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu   +18 more
wiley   +1 more source

Mixing weight determination for retrieving optical properties of polluted dust with MODIS and AERONET data

open access: yesEnvironmental Research Letters, 2016
In this study, an approach in determining effective mixing weight of soot aggregates from dust–soot aerosols is proposed to improve the accuracy of retrieving properties of polluted dusts by means of satellite remote sensing.
Kuo-En Chang   +7 more
doaj   +1 more source

Tables of Auxiliary Functions for the Nonconservative Rayleigh Phase Matrix in Semi-Infinite Atmospheres

open access: yesThe Astrophysical Journal Supplement Series, 1971
Tables of scattering functions and albedo for semiinfinite atmospheres according to nonconservative Rayleigh phase matrix for diffuse radiation ...
K. D. Abhyankar, A. L. Fymat
openaire   +1 more source

A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression

open access: yesMolecular Oncology, EarlyView.
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak   +8 more
wiley   +1 more source

Adaptaquin is selectively toxic to glioma stem cells through disruption of iron and cholesterol metabolism

open access: yesMolecular Oncology, EarlyView.
Adaptaquin selectively kills glioma stem cells while sparing differentiated brain cells. Transcriptomic and proteomic analyses show Adaptaquin disrupts iron and cholesterol homeostasis, with iron chelation amplifying cytotoxicity via cholesterol depletion, mitochondrial dysfunction, and elevated reactive oxygen species.
Adrien M. Vaquié   +16 more
wiley   +1 more source

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