Results 281 to 290 of about 179,498 (360)
Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation. [PDF]
Lugin ML +8 more
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American Journal of Hematology, Volume 101, Issue 7, Page 1516-1521, July 2026.
Jennifer A. Woyach +16 more
wiley +1 more source
Site-specific profiling of structure and function of Igµ B cell receptor glycans. [PDF]
Holborough-Kerkvliet MD +11 more
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Biomarkers for Precision Prognosis in Prostate Cancer: Imaging, Molecular, and Integrated Approaches. [PDF]
Khazaei Z, Pouliot F, Archambault L.
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Targetable Vulnerabilities in MYC-Driven B Cell Lymphomas Resistant to BCR Extinction. [PDF]
Brambillasca S +15 more
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How Epstein–Barr Virus Drives Multiple Sclerosis: New Mechanisms and Therapeutic Lessons
MedComm, Volume 7, Issue 7, July 2026.
Dan Liu, Wenhui Fan, Pengtao Jiao
wiley +1 more source
Left inferior vena cava - when an anatomical variation has surgical implications. [PDF]
Palmeira B +3 more
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Leukemia research : a Forum for Studies on Leukemia and Normal Hemopoiesis, 2020
Asciminib is a potent, orally bioavailable, investigational drug that specifically and potently inhibits the tyrosine kinase activity of native ABL1, together with that of the chimeric BCR-ABL1 oncoprotein which causes chronic myeloid leukemia (CML).
P. Manley, Louise Barys, S. Cowan-Jacob
semanticscholar +1 more source
Asciminib is a potent, orally bioavailable, investigational drug that specifically and potently inhibits the tyrosine kinase activity of native ABL1, together with that of the chimeric BCR-ABL1 oncoprotein which causes chronic myeloid leukemia (CML).
P. Manley, Louise Barys, S. Cowan-Jacob
semanticscholar +1 more source
Mechanisms of resistance to BCR–ABL TKIs and the therapeutic strategies: A review
Critical Reviews in Oncology/Hematology, 2015Liwu Fu
exaly +2 more sources

