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Correlation of bone age development with overweight and obesity in 23,305 children from Beijing. [PDF]
EndocrineZhou B +13 more
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Clinical Orthopaedics and Related Research, 1982
Bone matrix of 28-month-old (senescent) rats, compared with 13-month-old rats, is strikingly deficient in the capacity to induce bone formation. Similarly, implants of senescent matrix into one-month-old, rapidly-growing rats (weanlings) produce more bone than in 13-month-old adult recipients, but not nearly enough to compensate for deficiency in the ...
G T, Syftestad, M R, Urist
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Bone matrix of 28-month-old (senescent) rats, compared with 13-month-old rats, is strikingly deficient in the capacity to induce bone formation. Similarly, implants of senescent matrix into one-month-old, rapidly-growing rats (weanlings) produce more bone than in 13-month-old adult recipients, but not nearly enough to compensate for deficiency in the ...
G T, Syftestad, M R, Urist
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2009
This chapter will provide a general overview of the aging process followed by the potential effect that aging may have in bone biology. Three important aspects will be considered: decreased number of osteoblasts, increasing adipogenesis and significant osteoblast/osteocytes apoptosis during the aging process in bone.
Gimble, Jeffrey M. +3 more
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This chapter will provide a general overview of the aging process followed by the potential effect that aging may have in bone biology. Three important aspects will be considered: decreased number of osteoblasts, increasing adipogenesis and significant osteoblast/osteocytes apoptosis during the aging process in bone.
Gimble, Jeffrey M. +3 more
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Comprehensive Physiology, 2023
AbstractChanges in bone architecture and metabolism with aging increase the likelihood of osteoporosis and fracture. Age‐onset osteoporosis is multifactorial, with contributory extrinsic and intrinsic factors including certain medical problems, specific prescription drugs, estrogen loss, secondary hyperparathyroidism, microenvironmental and cellular ...
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AbstractChanges in bone architecture and metabolism with aging increase the likelihood of osteoporosis and fracture. Age‐onset osteoporosis is multifactorial, with contributory extrinsic and intrinsic factors including certain medical problems, specific prescription drugs, estrogen loss, secondary hyperparathyroidism, microenvironmental and cellular ...
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Baillière's Clinical Rheumatology, 1993
Bone can be divided into two kinds of tissue, cortical and trabecular bone. The skeleton comprises approximately 80% cortical bone, mainly in peripheral bones, and 20% trabecular bone, mainly in the axial skeleton. Bone density increases with skeletal growth to a peak in late adolescence or early adulthood.
P, Sambrook, P, Kelly, J, Eisman
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Bone can be divided into two kinds of tissue, cortical and trabecular bone. The skeleton comprises approximately 80% cortical bone, mainly in peripheral bones, and 20% trabecular bone, mainly in the axial skeleton. Bone density increases with skeletal growth to a peak in late adolescence or early adulthood.
P, Sambrook, P, Kelly, J, Eisman
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Pediatrics In Review, 1993
Skeletal maturation is expressed as bone age or skeletal age. Bone age is based on the orderly appearance of the ossification centers and, therefore, on the successive stages of the development (or maturity) of the skeleton as seen on radiographs. This is not a measure of size, but rather of shape and position.
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Skeletal maturation is expressed as bone age or skeletal age. Bone age is based on the orderly appearance of the ossification centers and, therefore, on the successive stages of the development (or maturity) of the skeleton as seen on radiographs. This is not a measure of size, but rather of shape and position.
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Current Pharmaceutical Biotechnology, 2017
The decline of tissue function in ageing is a consequence of many changes in the gene expression and other extrinsic factors. The molecular mechanisms underlying these changes are heavily investigated with focus on regulation of time-lapse gene expression.
Tilen, Kranjc +2 more
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The decline of tissue function in ageing is a consequence of many changes in the gene expression and other extrinsic factors. The molecular mechanisms underlying these changes are heavily investigated with focus on regulation of time-lapse gene expression.
Tilen, Kranjc +2 more
openaire +2 more sources