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Agglutination ofBordetellaspecies by lectins [PDF]
Phase I cells of Bordetella pertussis but not those of B. parapertussis, B. bronchiseptica or B. avium were agglutinated by Limulus polyphemus lectin. Most strains of B. pertussis but not those of the other species were also agglutinated by Helix pomatia lectin. In precipitation reactions between lectins and purified Bordetella lipopolysaccharide (LPS)
M J Corbel
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Bordetella holmesii: an under-recognised Bordetella species
Lancet Infectious Diseases, The, 2014Bordetella holmesii, first described in 1995, is believed to cause both invasive infections (bacteraemia, meningitis, endocarditis, pericarditis, pneumonia, and arthritis) and pertussis-like symptoms. Infection with B holmesii is frequently misidentified as being with B pertussis, the cause of whooping cough, because routine diagnostic tests for ...
Jacques Schrenzel +2 more
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Pathogenesis in Bordetella Species
Journal of Infectious Diseases, 1990P Novotny
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Isolation of Bordetella species from unusual infection sites
Introduction: Bordetella hinzii has been isolated mainly from respiratory specimens and from blood of immunocompromised patients, and Bordetella trematum from ear infections or leg, arm and ankle wounds and from diabetic foot ulcers. Bordetella holmesii is instead associated with bactaeremia in
Angela Famiglietti +1 more
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Bordetella Species Other than Bordetella pertussis
Clinical Microbiology Newsletter, 2015Most clinicians and microbiologists are familiar with Bordetella pertussis, its associated clinical syndromes and treatment with antimicrobials, and both biochemical and molecular methods for identification. Many of the other Bordetella species are much less well known, though they do cause clinical disease in humans.
Jocelyn A. Srigley +2 more
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All species of the genus bordetella contain genes for pertussis toxin of bordetella pertussis
Zentralblatt für Bakteriologie, Mikrobiologie und Hygiene. Series A: Medical Microbiology, Infectious Diseases, Virology, Parasitology, 1988A molecular probe for the PT-operon of B. pertussis hybridized with 4.7 Kb EcoRI-fragments of chromosomal DNAs of B. pertussis strain 475 phase I, phase IV, B. parapertussis strains 504 and 17903, B. bronchiseptica strain 214, B. parapertussis strain 17903-convertant of B. pertussis phage 134 but not with phage 134 DNA under stringent conditions of DNA-
M N, Rozinov +4 more
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Deoxyribonucleotide Sequence Relationships Among Bordetella Species [PDF]
Deoxyribonucleotide sequence relationships among currently recognized Bordetella species (Bordetella pertussis, Bordetella parapertussis, and Bordetella bronchiseptica) were examined by deoxyribonucleic acid (DNA) hybridization involving the hydroxyapatite batch procedures of Brenner and co-workers.
C R Manclark +2 more
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Environmental regulation of virulence factors in Bordetella species
BioEssays, 1993AbstractMany bacteria respond in a coordinate manner to environmental changes. External stimuli, sensed by receptors, are transduced to regulatory proteins which participate in well defined pathways of gene expression by varying their structure and mode of action. The network of environmental signal transduction is responsible for a fine and continuous
Scarlato V. +3 more
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Multiple species and isoforms of Bordetella pertussis toxin substrates
Biochemical and Biophysical Research Communications, 1988In purified G proteins from bovine brain cortex the ADP-ribosylated substrates of Bordetella pertussis toxin (PT) can be resolved in three polypeptides by polyacrylamide gel electrophoresis: a 39 kDa major substrate, corresponding to Go alpha and two others (40 and 41 kDa) assigned to alpha subunits of Gi-like proteins.
P, Brabet +6 more
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Genetic Regulation of Airway Colonization by Bordetella Species
American Journal of Respiratory and Critical Care Medicine, 1996Abstract Bordetella species are respiratory pathogens that infect humans and other animals. A majority of the virulence factors expressed by these bacteria are regulated by a master control locus, BvgAS, a member of the two-component family of signal transduction systems.
M H, Yuk, P A, Cotter, J F, Miller
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