Results 131 to 140 of about 17,249 (148)

C3 nephritic factor and mesangiocapillary glomerulonephritis

Pediatric Nephrology, 1997
The association of a C3 splitting activity, known as C3 nephritic factor (C3NeF), with mesangiocapillary glomerulonephritis (MCGN), especially MCGN type II, has long been known. Several forms of C3NeF are now recognised, the main one being an IgG which acts as an autoantibody binding to factor H, a normally occurring component of the complement system.
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[Mesangial C3-glomerulonephritis].

Vutreshni bolesti, 1991
12 patients with mesangial C3-glomerulonephritis, 7 women and 5 men, aged 16-42, mean age 29.7 years, were followed up for 1-18 years, mean time 5.7 years. The disease was manifested most often by macroscopic hematuria or low degree proteinuria. Single patients had arterial hypertension. In only one patient there was a nephrotic syndrome.
V, Dimitrova, V, Minkova, N, Belovezhdov
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Membranoproliferative Glomerulonephritis and C3 Glomerulopathy

2013
Membranoproliferative glomerulonephritis (MPGN) refers to a pattern of injury characterized by diffuse mesangial expansion due to mesangial and endocapillary proliferation and increased mesangial matrix, and thickened capillary walls, often with a double contour “tram-track” appearance [1, 2]. This pattern may be seen with immune complex deposition, or
Agnes B. Fogo, Arthur H. Cohen, Robert B. Colvin, J. Charles Jennette, Charles E. Alpers   +4 more
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C3 Glomerulopathy Masquerading as Acute Postinfectious Glomerulonephritis

American Journal of Kidney Diseases, 2012
We report the case of a 63-year-old man who presented with acute kidney injury, active urine sediment, nephrotic syndrome, and hypocomplementemia after a recent report of a sore throat. Kidney biopsy showed diffuse proliferative and exudative glomerulonephritis with C3-dominant staining by immunofluorescence.
Gagangeet, Sandhu, Anip, Bansal, Aditi, Ranade, James, Jones, Stanley, Cortell, Glen S, Markowitz   +5 more
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Membranoproliferative Glomerulonephritis and C3 Glomerulopathy

2014
The terms membranoproliferative glomerulonephritis (MPGN) and mesangiocapillary glomerulonephritis (MCGN) are interchangeable and refer to the light microscopic appearances of cellular proliferation in the mesangial regions of the glomeruli, with expansion of both cells and mesangial matrix, accompanied by thickening of the glomerular capillary walls ...
Daniel Gale, Mared Owen-Casey
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Membranoproliferative glomerulonephritis and C3 glomerulopathy

2015
Abstract Membranoproliferative glomerulonephritis (MPGN) is synonymous with mesangiocapillary glomerulonephritis and refers to light microscopic appearances of a kidney biopsy in which there are increased mesangial cells and matrix with thickening of the glomerular capillary walls, often with a double contour appearance.
Daniel P. Gale, Terry Cook
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Hypomorphic variant of C3, arthritis, and chronic glomerulonephritis

The Journal of Pediatrics, 1978
Decreased synthesis (hypomorphism) of the fast variant of the third component of complement was detected in three generations of a family in which the propositus has an immune complex-type glomerulonephritis, arthritis, and a false positive test for syphilis. An affected sibling has bursitis, hematuria, and proteinuria.
R H, McLean, A, Weinstein, I, Damjanov, N, Rothfield   +3 more
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Inherited C3 Deficiency With Recurrent Infections and Glomerulonephritis

Archives of Pediatrics & Adolescent Medicine, 1988
A 10-year-old Laotian boy had homozygous deficiency of the third component of complement and recurrent bacterial infections beginning at age 5 months. Cellular and humoral immunity were normal, as were polymorphonuclear leukocyte chemotaxis and bactericidal activities.
M S, Borzy, A, Gewurz, L, Wolff, D, Houghton, E, Lovrien   +4 more
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C3 Glomerulonephritis Triggered by Toxocariasis

Journal of the American Society of Nephrology, 2022
Mona Ben m'rad, Alexandre Beaulac, Steven Palanchuck, Veronique Beaunoyer, Me Linh Luong   +4 more
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C3 Nephritic Factor and Membranoproliferative Glomerulonephritis

1991
C3 nephritic factor (NeF) is an IgG autoantibody that is directed against neoantigenic determinants on the alternative pathway C3 convertase, C3bBb. It stabilizes the enzyme, thereby causing complement activation, and is characteristically associated with profound hypocomplementaemia. Clinically, NeF has striking associations with partial lipodystrophy
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