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MicroRNA-Mediated Regulatory Networks in <i>Helicobacter pylori</i>-Associated Gastric Cancer. [PDF]

Biochem Res Int
Sariani OK, Mohamadi M, Gharagheizi Z, Alaee M, Beig M, Sholeh M.   +5 more
europepmc   +1 more source

<i>Helicobacter pylori</i> in esophageal carcinogenesis: Potential culprit or forceful defender? [PDF]

Virulence
Ren D, Cui M, Yang D, Zhang Y, Hao Y, Yue Y, Wei B, Fan Q, Sun C, Li Q, Zhang M.   +10 more
europepmc   +1 more source

Novel CagA ELISA exhibits enhanced sensitivity of Helicobacter pylori CagA antibody

Novel CagA ELISA exhibits enhanced sensitivity of Helicobacter pylori CagA antibody
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Mixed Infection with cagA‐Positive and cagA‐Negative Strains of Helicobacter pylori

Helicobacter, 1996
ABSTRACT Background Helicobacter pylori infection has been implicated strongly in the pathogenesis of gastritis, peptic ulcer disease, gastric adenocarcinoma, and gastric lymphoma, but the reasons for these widely different clinical outcomes are unknown.
G T, Fantry, Q X, Zheng, P E, Darwin, A H, Rosenstein, S P, James   +4 more
openaire   +2 more sources

cagA+ Helicobacter pylori induces greater levels of prostaglandin E2 than cagA− strains

Prostaglandins & Other Lipid Mediators, 2004
cagA+ Helicobacter pylori (HP) infection is associated with an increased risk of distal gastric cancer. Previous studies investigating the effect of HP infection on prostaglandin E2 (PGE2) levels have not differentiated between cagA+ and cagA- strains and consequently have produced contradictory results.
Mohammed S, Al-Marhoon, Sheila, Nunn, Roger W, Soames   +2 more
openaire   +3 more sources

Oncogenic mechanisms of the Helicobacter pylori CagA protein

Nature Reviews Cancer, 2004
Infection with strains of Helicobacter pylori that carry the cytotoxin-associated antigen A (cagA) gene is associated with gastric carcinoma. Recent studies have shed light on the mechanism through which the cagA gene product, CagA, elicits pathophysiological actions.
Masanori Hatakeyama, Hatakeyama Masanori   +1 more
exaly   +3 more sources

CagA-mediated pathogenesis of Helicobacter pylori

Microbial Pathogenesis, 2016
Helicobacter pylori has been described as the main etiologic agent of gastric cancer, causing a considerable rate of mortality and morbidity in human population across the world. Although the infection mainly begins asymptomatically, but simply develops to peptic ulcer, chronic gastritis, lymphoma of the gastric mucosa and eventually adenocarcinoma ...
openaire   +5 more sources

Risk for Gastric Lymphoma in Persons with CagA⁺and CagA^‐Helicobacter pyloriInfection

The Journal of Infectious Diseases, 1997
Infection with Helicobacter pylori increases the risk for gastric non-Hodgkin's lymphoma (GNHL). Strains that express CagA protein are thought to be particularly virulent. It was determined whether CagA+ H. pylori infection increased the risk for GNHL more than CagA infection. Thirty-two cases and 130 controls previously tested for H. pylori antibodies
H L, Witherell, S, Hansen, E, Jellum, N, Orentreich, J H, Vogelman, J, Parsonnet   +5 more
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Serological differentiation of Helicobacter pylori CagA(+) and CagA(-) infections.

Archivum immunologiae et therapiae experimentalis, 2003
Many Helicobacterpylori strains causing gastroduodenal diseases have a cagA gene encoding CagA protein, a virulence factor of these bacteria. Anti-CagA antibodies produced by the majority of people infected with CagA(+) strains can indicate such an infection.
Magdalena, Chmiela, Monika, Wiśniewska, Leokadia, Bak-Romaniszyn, Tomasz, Rechciński, Izabela, Płaneta-Małecka, Władysław, Bielański, Stanisław J, Konturek, Małgorzata, Płonka, Magdalena, Klink, Wiesława, Rudnicka   +9 more
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Deregulation of β‐catenin signal by Helicobacter pylori CagA requires the CagA‐multimerization sequence

International Journal of Cancer, 2007
AbstractInfection with Helicobacter pylori cagA‐positive strains causes gastritis and peptic ulceration and is associated with gastric adenocarcinoma. The cagA gene product CagA is delivered into gastric epithelial cells, where it undergoes tyrosine phosphorylation by Src family kinases at the C‐terminal EPIYA‐repeat region.
Yo, Kurashima, Naoko, Murata-Kamiya, Kenji, Kikuchi, Hideaki, Higashi, Takeshi, Azuma, Satoshi, Kondo, Masanori, Hatakeyama   +6 more
openaire   +2 more sources