Results 161 to 170 of about 5,149,759 (252)

Aspirin use, lipoprotein(a), and calcific aortic valve disease: the Multi-ethnic Study of Atherosclerosis. [PDF]

Eur Heart J
Razavi AC, Bhatia HS, Marrero N, Dzaye O, Post WS, Nasir K, Tsai M, Vaccarino V, Vasan RS, Thanassoulis G, Tsimikas S, Sperling LS, Budoff MJ, Blumenthal RS, Blaha MJ, Whelton SP.   +15 more
europepmc   +1 more source

miR‐138‐5p Inhibits Aortic Valve Interstitial by Targeting SLC39A14 and Activating Nrf2 Signaling

Journal of Biochemical and Molecular Toxicology, Volume 40, Issue 9, September 2026.
Nrf2 Pathway Inhibition Attenuates miR‐138‐5p's Anti‐Calcification Effects in OM‐Induced hAVICs. A. ALP staining for ALP activity assessment in NC‐mimic, miR‐mimic, and miR‐mimic + ML385 groups. B. Alizarin Red staining for osteogenesis and calcium nIn osteogenic medium‐induced human aortic valve interstitial cells (hAVICs), miR‐138‐5p targets SLC39A14
Fei Yan, Ying Zhao, Yuanyuan Wu, Dubiao Xian, Lei Shi, Yunheng Liang, Ning Wang, Ruiling He, Hongwang Cui, Zhihao Xie   +9 more
wiley   +1 more source

Neurocardiology: Brain–Heart Interactions in the Heart

MedComm, Volume 7, Issue 9, September 2026.
The heart and brain maintain systemic homeostasis through bidirectional communication involving neural, biochemical, and mechanical pathways. Dysregulation of these pathways contributes to a spectrum of conditions involving heart–brain interactions, including Takotsubo syndrome, the epileptic heart, stroke–heart syndrome, heart failure, cardiac ...
Wentao Wang, Hao Jia, Han Han, Jiangping Song   +3 more
wiley   +1 more source

Calcific Aortic Valve Disease: Mechanism and Future Therapeutic Strategies. [PDF]

Cells
Hwang G, Jo S, Kwon H, Kwon M, Ku I, Song JK, Jo YH.   +6 more
europepmc   +1 more source

Patent Foramen Ovale: Epidemiology, Risk Factors, Pathophysiology, Clinical Features, Diagnosis, and Management

MedComm, Volume 7, Issue 9, September 2026.
The clinical manifestations of PFO. Although the majority of PFOs are benign and asymptomatic, they can present with a variety of clinical manifestations, or coexisting with other diseases and aggravating their symptoms, including cerebral diseases (such as CS, epilepsy, TIA, or migraine), systemic embolism (such as embolism in renal artery, coronary ...
Linlin Meng, Xiaotong Zhang, Guangrui Sun, Honggang Dai, Xiao Meng, Cheng Zhang, Yun Zhang   +6 more
wiley   +1 more source

Exploring CACNA1H as a potential candidate biomarker for calcific aortic valve disease. [PDF]

iScience
Chen J, Wang L, Li W, Li S, Duan X, Jiang S, Zhang Z, Zeng Q.   +7 more
europepmc   +1 more source

Calcific Aortic Valve Disease

, 2011
Jesper Hjortnaes, Elena Aikaw
openaire   +2 more sources

The Human PNPLA3I148M Polymorphism Alters the Renal Kinome and Transporters to Cause Hyperfiltration and Hypertension

FASEB BioAdvances, Volume 8, Issue 9, September 2026.
The present study used mice carrying the I148M polymorphism in the human Patatin‐like phospholipase domain‐containing protein 3 (PNPLA3I148M) to determine the role of this polymorphism in regulating blood pressure and renal filtration. Kinome analysis revealed that male PNPLA3I148M mice exhibited decreased phosphorylation of protein kinase c alpha ...
Gertrude Arthur, Michael I. Adenawoola, Sally Wahba, Bentley S. Montgomery, Claire B. Wingfield, Zachary A. Kipp, Terry D. Hinds Jr., David E. Stec   +7 more
wiley   +1 more source

Shared pathophysiological mechanisms between diabetes and calcific aortic valve disease: an immunometabolic perspective. [PDF]

Cardiovasc Diabetol
Yue Z, SiTu Q, Bian JH, Lu HX.
europepmc   +1 more source

In search of the smoking gun in calcific aortic valve disease [PDF]

The Journal of Thoracic and Cardiovascular Surgery, 2017
Takashi, Murashita, Vinay, Badhwar
openaire   +2 more sources