Results 31 to 40 of about 531,192 (386)

Calcific Myonecrosis

open access: yesacta orthopaedica et traumatologica turcica, 2008
Calcific myonecrosis is a rare complication of limb trauma, that may occur after many years. It is characterized by dystrophic calcification that develops in the late period following compartment syndrome usually in the lower limb. We present a 66-year-old man who developed calcific myonecrosis 35 years after surgical intervention for compartment ...
Melih, Güven   +5 more
openaire   +2 more sources

Multifocal cardiovascular calcification in patients with established cardiovascular disease; prevalence, risk factors, and relation with recurrent cardiovascular disease

open access: yesInternational Journal of Cardiology: Heart & Vasculature, 2020
Aims: The aim is to investigate (multifocal) cardiovascular calcification in patients with established cardiovascular disease (CVD), regarding prevalence, risk factors, and relation with recurrent CVD or vascular interventions.
Cilie C. van 't Klooster   +8 more
doaj   +1 more source

Influence of cell cycle phase on calcification in the coccolithophore Emiliania huxleyi [PDF]

open access: yes, 2008
Calcification of the cosmopolitan coccolithophore species Emiliania huxleyi was investigated in relation to the cell division cycle with the use of batch cultures.
Antia, Avan   +2 more
core   +1 more source

Chitosan/Hydroxyapatite Scaffolds with P28 as a Promising Osteoinductive Scaffold for Bone Healing Applications

open access: yesMicro, 2023
Despite bone’s inherent ability to heal, large bone defects remain a major clinical concern. This study proposes an off-the-shelf treatment combining chitosan/hydroxyapatite (CS/HAp) scaffolds, covalently linked with either bone morphogenetic protein-2 ...
Farah Alwani Azaman   +4 more
doaj   +1 more source

Study of the histomorphology of human pineal gland

open access: yesNational Journal of Clinical Anatomy, 2023
Introduction: Histologically, the pineal gland shows pinealocytes, glial cells, and sometimes calcific concretions. Age-related changes in these three components have been reported by various workers.
Gyanraj Singh   +3 more
doaj   +1 more source

Imaging correlates of molecular signatures in oligodendrogliomas. [PDF]

open access: yes, 2004
Molecular subsets of oligodendroglioma behave in biologically distinct ways. Their locations in the brain, rates of growth, and responses to therapy differ with their genotypes. Retrospectively, we inquired whether allelic loss of chromosomal arms 1p and
Betensky, Rebecca A   +10 more
core   +2 more sources

Role of Vascular Smooth Muscle Cell Phenotypic Switching and Calcification in Aortic Aneurysm Formation.

open access: yesArteriosclerosis, Thrombosis and Vascular Biology, 2019
Aortic aneurysm is a vascular disease whereby the ECM (extracellular matrix) of a blood vessel degenerates, leading to dilation and eventually vessel wall rupture.
Ploingarm Petsophonsakul   +9 more
semanticscholar   +1 more source

Long-term prognosis for individuals with hypertension undergoing coronary artery calcium scoring [PDF]

open access: yes, 2015
To examine the performance of coronary artery calcification (CAC) for stratifying long-term risk of death in asymptomatic hypertensive ...
Callister, Tracy Q   +14 more
core   +1 more source

The Role of Vascular Smooth Muscle Cells in Arterial Remodeling: Focus on Calcification-Related Processes

open access: yesInternational Journal of Molecular Sciences, 2019
Arterial remodeling refers to the structural and functional changes of the vessel wall that occur in response to disease, injury, or aging. Vascular smooth muscle cells (VSMC) play a pivotal role in regulating the remodeling processes of the vessel wall.
Armand M G Jaminon   +3 more
semanticscholar   +1 more source

Caspase-independent programmed cell death triggers Ca2PO4 deposition in an in vitro model of nephrocalcinosis [PDF]

open access: yes, 2018
We provide evidence of caspase-independent cell death triggering the calcification process in GDNF-silenced HK-2 ...
Anglani F.   +8 more
core   +1 more source

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