Results 191 to 200 of about 18,416,887 (403)
In Vivo Cell Fusion between Mesenchymal Stroma/Stem-Like Cells and Breast Cancer Cells [PDF]
Catharina Melzer +2 more
openalex +1 more source
The Effect of Dimethyl Sulfone on Gastrointestinal Cancer Cell Lines (AGS, HepG2 and KYSE-30) and Flow Cytometric Analysis of the Cell Cycle [PDF]
Background: Methylsulfonylmethane (MSM) is a dietary supplement to support healthy body. It is considered to be one of the least toxic substances in biology.
بهلولی, شهاب +2 more
core
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath +13 more
wiley +1 more source
Thoracic Surgery Committee on Adjuvant Therapy of Non-small Cell Lung Cancer
doaj +1 more source
Regulation of cancer cell metabolism
R. Cairns, I. Harris, T. Mak
semanticscholar +1 more source
YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
wiley +1 more source
Xiaoqiong Tang, Yaoying Long, Liuyue Xu, Xinyu Yan Department of Hematology, The First Affiliated Hospital of Chongqing Medical University, Chongqing City 400016, People’s Republic of ChinaCorrespondence: Xiaoqiong TangDepartment of Hematology, The
Tang X, Long Y, Xu L, Yan X
doaj
ErbB4‐mediated regulation of vasculogenic mimicry capability in breast cancer cells
Ryota Kawahara, Siro Simizu
openalex +1 more source
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source

