Results 201 to 210 of about 2,753,395 (319)
Mechanical Cell Reprogramming on Tissue-Mimicking Hydrogels for Cancer Cell Transdifferentiation. [PDF]
Ren X +13 more
europepmc +1 more source
STUDIES ON Zn65 UPTAKE BY EHRLICH ASCITES CANCER CELLS IN VITRO
SHYUNICHI FUJII
openalex +2 more sources
Effect of adriamycin and high-dose methotrexate chemotherapy onin vivo andin vitro cell-mediated immunity in cancer patients [PDF]
Jack A. Roth +2 more
openalex +1 more source
Using multi‐omic characterization, we aimed to identify key regulators specific to squamous cell lung carcinoma (SqCC). SqCC‐specific differentially expressed genes were integrated with metabolics data. High expression of the creatine transporter SLC6A8, along with elevated creatine levels, appeared to be a distinct metabolic feature of SqCC.
Johan Staaf +10 more
wiley +1 more source
Changes in the structuredness of cytoplasmic matrix (SCM) in human lymphocytes induced by PHA and cancer basic protein as measured in single cells [PDF]
Lea Čerček, Bojan Cercek
openalex +1 more source
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser +14 more
wiley +1 more source
Targeting cancer cell stiffness and metastasis with clinical therapeutics. [PDF]
Gajda AM, Rodríguez-López R, Er EE.
europepmc +1 more source
Cell cycle changes in ovarian cancer after arabinosylcytosine [PDF]
Patrick F. Sheehy +3 more
openalex +1 more source
Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu +18 more
wiley +1 more source

