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β-Catenin regulates expression of cyclin D1 in colon carcinoma cells
Nature, 1999Osamu Tetsu, Frank McCormick
exaly +2 more sources
Activating Wnt/&bgr;‐catenin signaling pathway for disease therapy: Challenges and opportunities
Pharmacology and Therapeutics, 2019&NA; Wnt/&bgr;‐catenin signaling pathway is essential for embryo development and adult tissue homeostasis and regeneration, abnormal regulation of the pathway is tightly associated with many disease types, suggesting that Wnt/&bgr;‐catenin signaling ...
Piao Huang +5 more
semanticscholar +1 more source
1999
Abstract Originally a-catenin was identified in immunoprecipitation experiments as a 102 kDa protein coprecipitating with antibodies against E-cadherin. Primary structure analysis of α-catenin revealed homology to vinculin, a protein first described as a component of focal adhesions, and suggested the existence of a vinculin-related ...
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Abstract Originally a-catenin was identified in immunoprecipitation experiments as a 102 kDa protein coprecipitating with antibodies against E-cadherin. Primary structure analysis of α-catenin revealed homology to vinculin, a protein first described as a component of focal adhesions, and suggested the existence of a vinculin-related ...
openaire +1 more source
WNT–β-catenin signalling — a versatile player in kidney injury and repair
Nature Reviews Nephrology, 2020Stefan J. Schunk +3 more
semanticscholar +1 more source
Melanoma-intrinsic β-catenin signalling prevents anti-tumour immunity
Nature, 2015S. Spranger, R. Bao, T. Gajewski
semanticscholar +1 more source
Science's STKE, 2001
Regulating the cytosolic concentration of the protein β-catenin is an important cell proliferation control mechanism. The cytoplasmic concentration of β-catenin remains low through an interaction with a protein complex consisting of adenomatous polyposis coli (APC), Axin, protein phosphatase 2A, and glycogen synthase kinase 3β (GSK3β ...
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Regulating the cytosolic concentration of the protein β-catenin is an important cell proliferation control mechanism. The cytoplasmic concentration of β-catenin remains low through an interaction with a protein complex consisting of adenomatous polyposis coli (APC), Axin, protein phosphatase 2A, and glycogen synthase kinase 3β (GSK3β ...
openaire +1 more source