Results 201 to 210 of about 24,232,284 (333)

Mechanical Cues Regulate Cargo Sorting and Export at the Golgi

open access: yesAdvanced Science, Volume 13, Issue 1, 5 January 2026.
A pathway is identified linking extracellular matrix stiffness to secretion via Src–FAK–AMPK–GBF1 signaling. Golgi‐localized GBF1 acts as a mechanotransducer whose phosphorylation controls post‐Golgi cargo sorting toward secretion or lysosomal degradation.
Greta Serafino   +21 more
wiley   +1 more source

Supplementary Fig. 5 from Hypoxia Attenuates Trastuzumab Uptake and Trastuzumab-Emtansine (T-DM1) Cytotoxicity through Redistribution of Phosphorylated Caveolin-1 [PDF]

open access: gold, 2023
Vineesh Indira Chandran   +14 more
openalex   +1 more source

Domain-Specific Partitioning of Uterine Artery Endothelial Connexin43 and Caveolin-1 [PDF]

open access: bronze, 2016
Bryan C. Ampey   +3 more
openalex   +1 more source

Plasma extracellular vesicles from APOE3 Christchurch carriers display a protective phenotype in early stages of autosomal dominant Alzheimer's disease

open access: yesAlzheimer's &Dementia, Volume 22, Issue 1, January 2026.
Abstract INTRODUCTION The PSEN1E280A mutation causes autosomal dominant Alzheimer's disease (ADAD) with predictable onset, enabling presymptomatic studies. Extracellular vesicles (EVs) are emerging biomarkers of cognitive decline, but their role in early ADAD is unclear.
Lina Pineda‐Lopez   +14 more
wiley   +1 more source

Nanobiotechnology‐Based Approaches for Targeted Glioma Therapy

open access: yesAdvanced NanoBiomed Research, Volume 6, Issue 1, January 2026.
Nanoparticle systems cross the blood–brain barrier to deliver drugs precisely to glioma cells. Lipid, polymeric, and magnetic carriers enable controlled release, tumor targeting, and microenvironment modulation. These nanoplatforms enhance chemotherapy, radiotherapy, and immunotherapy, improving therapeutic efficacy while reducing systemic toxicity ...
Jinwei Li   +11 more
wiley   +1 more source

Tumor Electric Field Therapy Inhibits TGF‐β/C1R Signaling Axis‐Driven Epithelial‐Mesenchymal Transition in Glioblastoma

open access: yesCNS Neuroscience &Therapeutics, Volume 32, Issue 1, January 2026.
Tumor electric field therapy (TEFT) inhibits glioblastoma progression by suppressing the TGF‐β/SMAD2/3/STAT3/C1R axis to reverse epithelial‐mesenchymal transition (EMT). C1R knockdown reduces tumor growth and mesenchymal characteristics. This study identifies C1R as a key molecular target of TEFT and a potential biomarker for the aggressive mesenchymal
Junyi Chen   +12 more
wiley   +1 more source

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