Results 101 to 110 of about 2,002 (125)

Integrating exposomics into biomedicine. [PDF]

Science
Miller GW, Banbury Exposomics Consortium.   +1 more
europepmc   +1 more source

Age-related changes in the proteome and mitochondrial metabolism of rabbit adipose-derived stromal/stem cells. [PDF]

Sci Rep
Toto Nienguesso A, Jung JS, Alfes M, Schindler M, Täubert L, Schmidt C, Navarrete Santos A.   +6 more
europepmc   +1 more source

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The mechanism and biomarker function of Cavin-2 in lung ischemia-reperfusion injury

Computers in Biology and Medicine, 2022
Lung Ischemia Reperfusion injury(LIRI) is one of the most predominant complications of ischemic lung disease. Cavin-2 emerged as a regulator of a variety of cellular processes, including endocytosis, lipid homeostasis, signal transduction and tumorigenesis, but the function of Cavin-2 in LIRI is unknown.
Sun Linao
exaly   +3 more sources

Cavin-2 is a specific marker for detection of well-differentiated liposarcoma

Biochemical and Biophysical Research Communications, 2017
Caveolae are cholesterol enriched invaginations of the plasma membrane involved in a variety of processes, including glucose and fatty acids absorption, cell transduction and mechanoprotection. The biogenesis and function of caveolae depend on the activity of Caveolin (Cav-1, -2 and -3) and Cavin (Cavin-1, -2, -3 and -4) protein families.
Alessandro Fanzani, Marika Vezzoli, Manuela Cominelli   +2 more
exaly   +5 more sources

Loss of Cavin-2 destabilizes phosphatase and tensin homologue and enhances Akt signalling pathway in cardiomyocytes

Cardiovascular Research
Abstract Aims Specific cavins and caveolins, known as caveola-related proteins, have been implicated in cardiac hypertrophy and myocardial injury. Cavin-2 forms complexes with other caveola-related proteins, but the role of Cavin-2 in cardiomyocytes (CMs) is poorly understood. Here, we investigated an
Takehiro Ogata, Satoaki Matoba, Yumika Tsuji   +2 more
exaly   +3 more sources

Cavin‐2 in oral cancer: A potential predictor for tumor progression

Molecular Carcinogenesis, 2015
Cavin‐2 (CVN2) affects formation of large caveolae, which are membrane‐rich cholesterol domains associated with several functions in signal transduction. Accumulating evidence suggests that CVN2 is present in many cellular types; however, the molecular mechanisms of CVN2 in cancers and its clinical relevance are unknown.
Motoharu, Unozawa, Atsushi, Kasamatsu, Morihiro, Higo, Chonji, Fukumoto, Tomoyoshi, Koyama, Tomomi, Sakazume, Dai, Nakashima, Katsunori, Ogawara, Hidetaka, Yokoe, Masashi, Shiiba, Hideki, Tanzawa, Katsuhiro, Uzawa   +11 more
openaire   +2 more sources

Abstract 301: Cavin-2 Deletion Attenuates Fibroblast Activation and Cardiac Fibrosis via TGF-β Signaling by Interacting With Hsp90

Circulation Research, 2020
Introduction: Heart failure (HF) is a progressive disease associated with high morbidity and mortality. A major cause of HF is the adverse tissue remodeling with interstitial fibrosis. Excessive extracellular matrix (ECM) accumulation is involved in the poor outcome.
Yusuke Higuchi, Takehiro Ogata, Naohiko Nakanishi, Akira Sakamoto, Yumika Tsuji, Shinya Tomita, Satoaki Matoba   +6 more
openaire   +1 more source

Allergic asthma induce opposite changes in the expression of caveolin-1 and cavin-2 in airway and vascular pulmonary smooth muscle

European Respiratory Journal, 2014
Introduction Caveolin-1 (Cav-1) and Cavin-2 are fundamental signalling scaffold proteins involved in airway smooth muscle (ASM) contraction; however few is known about their expression in asthma. Objective The expression of Cav-1 and Cavin-2 in an asthma model was evaluated.
Sandra Luz Sánchez Hernández, Mayra Álvarez Santos, Fernando Gutiérrez Aguilar, Fernando Ávila Carbajal, Norma Bobadilla, Ricardo Lascurain, Gabriela Gutiérrez Olguín, Blanca Bazán Perkins   +7 more
openaire   +1 more source

Abstract 520: Cavin-2/SDPR in Cardiac Fibroblasts Modulates TGF-β/Smad Signaling and Promotes Pressure Overload-induced Fibrosis

Circulation Research, 2019
Introduction: Heart failure (HF) is a debilitating disease associated with high morbidity and mortality. The high mortality rate reflects the inadequacy of modern therapy and calls for new treatments. A major cause of HF is the adverse tissue remodeling with fibrosis. Excessive extracellular matrix (ECM) turnover is involved in
Yusuke Higuchi, Takehiro Ogata, Naohiko Nakanishi, Masahiro Nishi, Akira Sakamoto, Yumika Tsuji, Satoaki Matoba   +6 more
openaire   +1 more source

P151 L’hypoxie inhibe l’expression de Cavin1/PTRF et Cavin-2/SDPR et induit la disparition des cavéoles dans les adipocytes

Diabetes & Metabolism, 2014
Introduction Lors de l’obesite le developpement de zones d’hypoxie dans le tissu adipeux est implique dans le developpement de la resistance a l’insuline en inhibant la voie de signalisation de l’insuline dans les adipocytes. L’activation du recepteur de l’insuline depend de sa localisation dans les caveoles, dont la formation et la fonction sont ...
C. Regazzetti, K. Dumas, S. Lacas-Gervais, P. Peraldi, I. Dugail, S. Le Lay, Y. Le Marchand-Brustel, J. Tanti, M. Cormont, S. Giorgetti-Peraldi   +9 more
openaire   +1 more source