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Blocking cell death to enhance cell death
Science Translational Medicine, 2017Inhibiting caspase activation during apoptosis may enhance the ability of the immune system to target cancer cells.
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Molecular Cell
Cells can undergo death through various genetically regulated pathways, each leading to distinct tissue outcomes. Emerging evidence suggests that these pathways are molecularly interconnected, indicating that the mode of death is determined not only by apical signals but by the availability of terminal executioners/substrates.
Hamid, Kashkar, Manolis, Pasparakis
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Cells can undergo death through various genetically regulated pathways, each leading to distinct tissue outcomes. Emerging evidence suggests that these pathways are molecularly interconnected, indicating that the mode of death is determined not only by apical signals but by the availability of terminal executioners/substrates.
Hamid, Kashkar, Manolis, Pasparakis
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European Heart Journal, 1983
Irrespective of age or species, injured cardiac myocytes accumulate Ca2+. In this paper four different aspects of the Ca2+-overloading phenomenon are discussed. These aspects include the conditions under which it occurs, the possible routes of Ca2+ entry, the metabolic consequences of the raised tissue Ca2+ and possible protective procedures.
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Irrespective of age or species, injured cardiac myocytes accumulate Ca2+. In this paper four different aspects of the Ca2+-overloading phenomenon are discussed. These aspects include the conditions under which it occurs, the possible routes of Ca2+ entry, the metabolic consequences of the raised tissue Ca2+ and possible protective procedures.
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Journal of Neurobiology, 1992
AbstractExcitotoxicity refers to the ability of glutamate or related excitatory amino acids to mediate the death of central neurons under certain conditions, for example, after intense exposure. Such excitotoxic neuronal death may contribute to the pathogenesis of brain or spinal cord injury associated with several human disease states.
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AbstractExcitotoxicity refers to the ability of glutamate or related excitatory amino acids to mediate the death of central neurons under certain conditions, for example, after intense exposure. Such excitotoxic neuronal death may contribute to the pathogenesis of brain or spinal cord injury associated with several human disease states.
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A guide to cell death pathways
Nature Reviews Molecular Cell Biology, 2023Junying Yuan +2 more
exaly
1991
Cell death during the ontogeny of the nervous system allows for competitive innervation of targets (Hamburger and Oppenheim, 1982; Levi-Montalcini, 1987). Neurons that successfully compete for target-derived trophic factors, such as NGF, at critical stages in development are spared cell death. Exogenous NGF, or its withdrawal by anti-NGF, has permanent
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Cell death during the ontogeny of the nervous system allows for competitive innervation of targets (Hamburger and Oppenheim, 1982; Levi-Montalcini, 1987). Neurons that successfully compete for target-derived trophic factors, such as NGF, at critical stages in development are spared cell death. Exogenous NGF, or its withdrawal by anti-NGF, has permanent
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Programmed Cell Death or Cell Death Programme? That is the Question
Journal of Theoretical Biology, 2001D, Ratel +4 more
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Classification of cell death: recommendations of the Nomenclature Committee on Cell Death
Cell Death & Differentiation, 2005Kroemer, G. +12 more
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