Asxl1 loss in mice leads to microcephaly by regulating neural stem cell survival. [PDF]
Anim Cells Syst (Seoul)Kim H, Kim AR, Byun S, Um SJ.
europepmc +1 more source
Differing survival of normal and sensitised spleen cells transferred to allogeneic hosts
, 1965 B. G. Hauler, M. Schlesinger, D. B. Amos
openalex +1 more source
Proceedings: The relationship between cell survival, chromosome aberrations and DNA repair in tumour cell lines of differential sensitivity to X-rays and sulphur mustard [PDF]
, 1974 David Scott, Margaret Fox, B.W. Fox
openalex +1 more source
Negative Regulation of PKB/Akt-Dependent Cell Survival by the Tumor Suppressor PTEN
Cell, 1998 V. Stambolic +9 more
semanticscholar +1 more source
Ubiquitination of transcription factors in cancer: unveiling therapeutic potential
Molecular Oncology, EarlyView.In cancer, dysregulated ubiquitination of transcription factors contributes to the uncontrolled growth and survival characteristics of tumors. Tumor suppressors are degraded by aberrant ubiquitination, or oncogenic transcription factors gain stability through ubiquitination, thereby promoting tumorigenesis.
Dongha Kim, Hye Jin Nam, Sung Hee Baek
wiley +1 more source
Effects of Blue Light on the Survival of UV-irradiated Fern Gametophytic Cells
, 1967 Yukio Kato
openalex +2 more sources
TSC2 Mediates Cellular Energy Response to Control Cell Growth and Survival
Cell, 2003 K. Inoki, Tianqing Zhu, K. Guan
semanticscholar +1 more source
Targeted protein degradation in oncology: novel therapeutic opportunity for solid tumours?
Molecular Oncology, EarlyView.Current anticancer therapies are limited by the occurrence of resistance and undruggability of most proteins. Targeted protein degraders are novel, promising agents that trigger the selective degradation of previously undruggable proteins through the recruitment of the ubiquitin–proteasome machinery. Their mechanism of action raises exciting challenges,
Noé Herbel, Sophie Postel‐Vinay
wiley +1 more source
A COMPARISON OF SURVIVAL AND REPAIR OF UV-INDUCED DNA DAMAGE IN CULTURED INSECT VERSUS MAMMALIAN CELLS [PDF]
, 1977 Thomas M. Koval +3 more
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Molecular Oncology, EarlyView.Nuclear prothymosin α inhibits epithelial‐mesenchymal transition (EMT) in lung cancer by increasing Smad7 acetylation and competing with Smad2 for binding to SNAI1, TWIST1, and ZEB1 promoters. In early‐stage cancer, ProT suppresses TGF‐β‐induced EMT, while its loss in the nucleus in late‐stage cancer leads to enhanced EMT and poor prognosis.
Liyun Chen +12 more
wiley +1 more source