Results 151 to 160 of about 98,361 (208)
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The Journal of Nervous and Mental Disease, 1977
Great strides have been made in understanding the pathogenesis of cerebral edema. Treatment is usually successful, particularly with the newer modes of management (mannitol, steroids, hyperventilation); however, cerebral edema is occasionally resistant to all modes of therapy.
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Great strides have been made in understanding the pathogenesis of cerebral edema. Treatment is usually successful, particularly with the newer modes of management (mannitol, steroids, hyperventilation); however, cerebral edema is occasionally resistant to all modes of therapy.
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Neurosurgery, 1986
Acute mountain sickness (AMS) is usually a benign and self-limited illness that befalls previously healthy individuals who ascend rapidly to high altitude without sufficient acclimatization. In its more severe forms, AMS can progress to a life-threatening condition in which pulmonary or cerebral edema can occur singly or in concert.
Allan J. Hamilton +2 more
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Acute mountain sickness (AMS) is usually a benign and self-limited illness that befalls previously healthy individuals who ascend rapidly to high altitude without sufficient acclimatization. In its more severe forms, AMS can progress to a life-threatening condition in which pulmonary or cerebral edema can occur singly or in concert.
Allan J. Hamilton +2 more
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Seminars in Nephrology, 2001
Two major types of brain edema may be discriminated, characterized by intra- or extracellular fluid accumulation. Intracellular (cytotoxic) edema is found after cerebral ischemia, trauma, intoxications, and metabolic disorders. Pathogenetic mechanisms include (1) failure of active Na+ export via Na/K-ATPase because of energy shortage, (2) increased Na+-
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Two major types of brain edema may be discriminated, characterized by intra- or extracellular fluid accumulation. Intracellular (cytotoxic) edema is found after cerebral ischemia, trauma, intoxications, and metabolic disorders. Pathogenetic mechanisms include (1) failure of active Na+ export via Na/K-ATPase because of energy shortage, (2) increased Na+-
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Cerebral edema associated with meningiomas
Surgical Neurology, 1987Fifty patients with intracranial meningiomas have been retrospectively examined, and the cerebral edema on computed tomography scan has been correlated with the clinical evolution, size, location, and histological features of the tumor. The degree of brain edema was found to be related to the clinical evolution and the size of the tumor, whereas the ...
MAIURI F +6 more
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Neurosurgery Clinics of North America, 1994
Cerebral edema continues to plague clinicians caring for patients with acute catastrophic neurologic disease. The defect responsible for the accumulation of water in the brain appears to reflect loss of the strict permeability barrier of the cerebral vasculature.
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Cerebral edema continues to plague clinicians caring for patients with acute catastrophic neurologic disease. The defect responsible for the accumulation of water in the brain appears to reflect loss of the strict permeability barrier of the cerebral vasculature.
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Cerebral Edema Associated with Meningiomas
Neurosurgery, 1983Abstract The cerebral edema, as judged by computed tomographic scan, associated with supratentorial meningiomas was assessed in 55 cases. No relationship to the occurrence or the degree of edema could be established with respect to meningioma location, histological type, tumor vascularity, cellularity, number of mitotic figures, necrosis,
R K Coates +3 more
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The pathology of cerebral edema
Human Pathology, 1974Abstract In a review of the literature dealing with cerebral edema the author analyzes concepts, pathogenetic mechanisms, and the recent classification into vasogenic and cytotoxic forms. The relationship between systemic and cerebral edema is discussed, as well as the effects of hydrocephalus.
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1999
The blood-brain barrier (BBB) which is located in the continuous endothelial lining of cerebral blood vessels rigidly controls exchange of water soluble compounds under physiological conditions. Under pathological conditions such as trauma or ischemia, BBB permeability may increase thus allowing plasma constituents to escape into brain tissue.
Lothar Schilling, Michael Wahl
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The blood-brain barrier (BBB) which is located in the continuous endothelial lining of cerebral blood vessels rigidly controls exchange of water soluble compounds under physiological conditions. Under pathological conditions such as trauma or ischemia, BBB permeability may increase thus allowing plasma constituents to escape into brain tissue.
Lothar Schilling, Michael Wahl
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Cerebral Edema in Diabetic Ketoacidosis
New England Journal of Medicine, 1985Cerebral edema causes or contributes to death in diabetic ketoacidosis under two distinct circumstances — in comatose patients and during treatment in conscious patients.
David A. Simmons +2 more
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Cerebral Edema and Diabetic Ketoacidosis
Annals of Internal Medicine, 1982Excerpt To the editor: We write to comment on the article by Fein and associates (1). From information reported by Clements and colleagues (2), which confirmed our own observations, we suspect that...
Patricia Carroll, Robert Matz
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