Results 211 to 220 of about 260,357 (315)

Enhanced chemotaxis and degradation of nonylphenol in Pseudoxanthomonas mexicana via CRISPR-mediated receptor modification. [PDF]

open access: yesSci Rep
Chai R   +12 more
europepmc   +1 more source

Neutrophil Chemotaxis Dysfunction in Human Periodontitis [PDF]

open access: bronze, 1980
Thomas E. Van Dyke   +3 more
openalex   +1 more source

Spatiotemporal Adaptations‐Driven Dynamic Thra Activation Simulates a Skin Wound Healing Response

open access: yesAdvanced Science, EarlyView.
This study uncovers the thyroid hormone receptor Thra as a pivotal regulator that spatiotemporally orchestrates dual‐phase skin repair. Epidermal Thra accelerates barrier restoration via glutathione‐driven keratin assembly, while dermal Thra establishes angiogenic niches through SAA3‐FN1‐mediated matrix remodeling.
Zeming Li   +15 more
wiley   +1 more source

Monoallelic mutations in MMD2 cause autosomal dominant aggressive periodontitis. [PDF]

open access: yesJ Exp Med
Iwata T   +33 more
europepmc   +1 more source

PLAUR+ Neutrophils Drive Anti‐PD‐1 Therapy Resistance in Patients with Hepatocellular Carcinoma by Shaping an Immunosuppressive Microenvironment

open access: yesAdvanced Science, EarlyView.
The present study reveals that PLAUR+ neutrophils are enriched in immunotherapy non‐responders and correlate with poor prognosis. PLAUR+ neutrophils dictate immunotherapy resistance in hepatocellular carcinoma by forming an immunosuppressive tumor microenvironment characterized by CD8+ T cell exclusion and macrophage‐dependent immune suppression.
Shaoqing Liu   +11 more
wiley   +1 more source

Interplay between chemotaxis, quorum sensing, and metabolism regulates Escherichia coli-Salmonella Typhimurium interactions in vivo. [PDF]

open access: yesPLoS Pathog
Laganenka L   +9 more
europepmc   +1 more source

Sensory adaptation in bacterial chemotaxis: regulation of demethylation [PDF]

open access: bronze, 1985
Marilyn R. Kehry   +2 more
openalex   +1 more source

Cancer Cell‐Derived Large Extracellular Vesicles Promote Venous Thromboembolism by Activating NETosis Through Delivering CYBA

open access: yesAdvanced Science, EarlyView.
Venous thromboembolism (VTE) is a major threat to cancer patients. Cancer‐derived large extracellular vesicles deliver CYBA (p22phox) to neutrophils, triggering reactive oxygen species (ROS) and citrullinated histone H3 (citH3) formation. This drives neutrophil extracellular traps formation (NETosis), elevating VTE risk.
Xiangji Li   +8 more
wiley   +1 more source

The minimal chemotactic cell. [PDF]

open access: yesSci Adv
Borges-Fernandes B   +11 more
europepmc   +1 more source

Home - About - Disclaimer - Privacy