Some studies on the chemical modification of ϵ-toxin of Clostridium perfringens type D
Biochimica et Biophysica Acta, 1963Abstract Chemical modification of ϵ-toxin of Clostridium perfringens type D by acetylation and guanidination has been carried out. With both reagents when 50% of the free amino groups were reacted the toxicity dropped to 7%. Further modification of the free amino groups by guanidination reduced the toxicity to 0.6% and with acetylation the toxicity
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Vitamin E-Enhanced Humoral Antibody Response to Clostridium Perfringens Type D in Sheep
British Veterinary Journal, 1983SUMMARY Dietary supplementation of vitamin E ([dl]-α-tocopheryl acetate) significantly increased humoral antibody production in sheep vaccinated with Clostridium perfringens type C and D toxoids, as measured in an ELISA test. A vitamin E adjuvant increased antibody production much more than dietary supplementation.
R P, Tengerdy +4 more
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Clostridium perfringens type D epsilon intoxication in one‐day‐old calves
Veterinary Record, 2009Clostridium perfringens type D enterotoxaemia, or ‘pulpy kidney disease’, is a very common cause of sudden death in sheep worldwide. There are numerous scientific publications on the epidemiology and pathology of enterotoxaemia in sheep; in contrast, there have been few studies of type ...
P J, Watson, S F E, Scholes
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Genomic analysis of Clostridium perfringens type D isolates from goat farms
Veterinary MicrobiologyC. perfringens type D strains are the leading cause of enterotoxaemia in ruminants such as goats, sheep, and cattle. However, there has been no prior research on the genomic characteristics of C. perfringens type D strains from various regions in China.
Hang, Feng +9 more
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Ultrastructural changes in the brain of mice given Clostridium perfringens type D epsilon toxin
Journal of Comparative Pathology, 1984Mice were given lethal and sublethal doses of Clostridium perfringens Type D epsilon toxin and the early morphological changes in perfusion-fixed intoxicated brains were examined from 30 min to 6 h post-inoculation. The initial ultrastructural finding was swelling of astrocytes, especially the perivascular extensions of these cells.
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Pathogenicity and virulence of Clostridium perfringens
Virulence, 2021Iman Mehdizadeh Gohari +2 more
exaly
Enterotoxemia (Clostridium perfringens, Type D) Toxoids
Veterinary Clinics of North America: Food Animal Practice, 1990openaire +1 more source
CD31 (PECAM-1) Serves as the Endothelial Cell-Specific Receptor of Clostridium perfringens β-Toxin
Cell Host and Microbe, 2020Marianne Wyder +2 more
exaly
Enterotoxaemia caused by Clostridium perfringens Type D in farmed fallow deer
Australian Veterinary Journal, 1985openaire +2 more sources
Expansion of the Clostridium perfringens toxin-based typing scheme
Anaerobe, 2018Julian I Rood, Jake A Lacey, Dena Lyras
exaly