Results 41 to 50 of about 21,356 (260)
Interleukin‐18 signaling promotes activation of hepatic stellate cells in mouse liver fibrosis
Hepatology, EarlyView., 2022 Interleukin‐18 signaling promotes activation of hepatic stellate cells in mouse liver fibrosis. Abstract Background and Aims Nucleotide‐binding oligomerization domain‐like receptor‐family pyrin domain‐containing 3 (NLRP3) inflammasome activation has been shown to result in liver fibrosis.
Jana Knorr +19 more
wiley +1 more source
Indonesian Journal of Obstetrics and Gynecology, 2013 Objective: To analyze correlation between concentration of estrogen receptor and imunoexpression of Col3A1 gene on uterosacral ligament of postmenopausal uterine prolapse patient.
Erick C. Asmara
doaj +1 more source
COL3A1 and SNAP91: novel glioblastoma markers with diagnostic and prognostic value
Oncotarget, 2016 Although patients with glioblastoma (GBM) have grave prognosis, significant variability in patient outcome is observed. This study aims to identify novel targets for GBM diagnosis and therapy. Microarray data (GSE4290, GSE7696, and GSE4412) obtained from the Gene Expression Omnibus was used to identify the differentially expressed genes (DEGs) by ...
Gao, Yuan-Feng +11 more
openaire +3 more sources
The MFs develop normally in Col3a1 mutant mice.
, 2013 Double IHC of Zic (red) and Reelin (green) (A–D) or Tuj1 (green) (E–H) in Col3a1+/+ and Col3a1−/− brains revealed a normal distribution of Zic+ cells in all brains analyzed. (I and J) Laminin staining of primary MFs revealed no differences between Col3a1+
Rong Luo (187167) +4 more
core +1 more source
Col3a1−/− mice have cortical abnormalities.
, 2013 Sagittal sections from one Col3a1+/+ (A) and three Col3a1−/− forebrains (B–I) stained with Nissl. In contrast to the well-developed cortex in Col3a1+/+ brains (A), cortical malformation was seen in Col3a1−/− brains, characterized by the presence of ...
Rong Luo (187167) +4 more
core +1 more source
, 2022 One of the earliest signs of dysregulation of the homeostatic process of fibrosis, associated with pathology in chronic conditions such as rheumatoid arthritis (RA), is the overexpression of collagen type III (COL-3).
Camargo da Rosa, Larissa +6 more
core +1 more source
Epistasis of polymorphisms related to the articular cartilage extracellular matrix in knee osteoarthritis: Analysis-based multifactor dimensionality reduction [PDF]
Genetics and Molecular Biology, 2020 Osteoarthritis (OA) is a complex disease with a multifactorial etiology. The genetic component is one of the main associated factors, resulting from interactions between genes and environmental factors.
Javier Fernández-Torres +5 more
doaj +1 more source
Gene expression of COL1A1 and COL3A1.
, 2013 COL1A1 and CoL3A1 mRNA normalized to tissue weight, presented as fold changes in the exercise group (filled bars) relative to the mean of the control group (open bars) in Achilles tendon and soleus muscle.
Andreas Kjaer (296549) +5 more
core +1 more source
Frontiers in Cardiovascular Medicine, 2022 BackgroundVascular Ehlers-Danlos syndrome (vEDS) is a rare inherited connective tissue disorder due to pathogenic variants in COL3A1 leading to medium-size-artery (MSA) dissection, aneurysm, rupture. Aortic lesions are rarer and less investigated.
Salma Adham +17 more
doaj +1 more source
COL3A1, CXCL8, VCAN, THBS2, and COL1A2 are correlated with the onset of biliary atresia
Medicine, 2023 Background: Biliary atresia (BA) is a devastating progressive fibro inflammatory disorder in infants. The exact etiology of BA is still unclear. This study aimed screen key genes potentially associated with the occurrence of BA.
Hui Li, Lei Cao, Hong Li
openaire +2 more sources