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Digestion, 1998
Research in hereditary forms of colorectal cancer (CRC) has increased almost logarithmically thanks in a major way to momentous discoveries in molecular genetics during the past decade. Between 10 and 20% of the total CRC burden is due to Mendelian-inherited CRC syndromes. The paradigm for hereditary CRC is familial adenomatous polyposis (FAP), wherein
H T, Lynch, J F, Lynch
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Research in hereditary forms of colorectal cancer (CRC) has increased almost logarithmically thanks in a major way to momentous discoveries in molecular genetics during the past decade. Between 10 and 20% of the total CRC burden is due to Mendelian-inherited CRC syndromes. The paradigm for hereditary CRC is familial adenomatous polyposis (FAP), wherein
H T, Lynch, J F, Lynch
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Surgical Clinics of North America, 1983
To manage recurrent colorectal cancer effectively, the surgeon must be aware of the prognostic significance of the pathologic features of the primary disease, the adequacy of the original operation, the time since the initial treatment, and the various kinds of recurrence.
H C, Polk, J S, Spratt
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To manage recurrent colorectal cancer effectively, the surgeon must be aware of the prognostic significance of the pathologic features of the primary disease, the adequacy of the original operation, the time since the initial treatment, and the various kinds of recurrence.
H C, Polk, J S, Spratt
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Pentastomiasis and cancer of the colon
Transactions of the Royal Society of Tropical Medicine and Hygiene, 1978Abstract The case is described of a 50-year-old man, admitted to a small mission hospital in Zaire, where a preliminary diagnosis of cancer was made. On referral to a bigger hospital he was found to be infected with Entamoeba histolytica and biopsy of the peritoneal tumour revealed a pentastomid nymph, probably of Armillifer armillatus, the most ...
I C, Bygbjerg, M P, Rask
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2012
Accumulation of genetic and epigenetic alterations transforms normal colonic epithelial cells to adenocarcinoma cells. Genetic alterations include mutations in tumor suppressor genes and oncogenes, whereas epigenetic mechanisms are defined as heritable alterations in gene expression that is independent of changes in the primary DNA sequence.
Sharad, Khare, Mukesh, Verma
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Accumulation of genetic and epigenetic alterations transforms normal colonic epithelial cells to adenocarcinoma cells. Genetic alterations include mutations in tumor suppressor genes and oncogenes, whereas epigenetic mechanisms are defined as heritable alterations in gene expression that is independent of changes in the primary DNA sequence.
Sharad, Khare, Mukesh, Verma
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Chemoprevention of Colon Cancer
2001Adenocarcinoma of the large bowel presents a major public health problem. It affects about 1 person in 20 in the United States and Western countries [1]. It is the second most common cause of cancer death in American males, and the third most common cause in American females.
M, Mulcahy, A, Benson
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