Results 261 to 270 of about 383,954 (303)

Colorectal Neoplasms

2014
Abstract Colorectal cancer is primarily a disease of urban, industrialized societies. In the United States, the lifetime risk for the development of this cancer is approximately 6%. Recent data have suggested that the incidence rates for colorectal cancer may be decreasing gradually in some subgroups of the population.
openaire   +1 more source

Nonprotruding Colorectal Neoplasms: Epidemiologic Viewpoint

World Journal of Surgery, 2000
AbstractHistologic sections from 781 nonprotruding colorectal neoplasias (adenomas and early carcinomas) recorded in Sweden and in Japan were reviewed by the same observer, using strict histologic criteria. Low grade dysplasia (LGD) was present in 82.8% (299/361) of the nonprotruding neoplasms recorded in Sweden but in only 42.6% (179/420) of those ...
openaire   +2 more sources

Prevention and Chemoprevention of Colorectal Neoplasms

Digestive and Liver Disease, 2002
Main purpose of the review is to analyse the impact of the current approaches for colorectal cancer prevention, including chemoprevention. Available evidence does not support the contention that a more appropriate diet can be of great help in the prevention of these neoplasms, either because the scientific evidence is poor and highly controversial, or ...
openaire   +3 more sources

Detecting Colorectal Neoplasms

Journal of Clinical Gastroenterology, 1987
Because of the controversy surrounding the detection of colorectal neoplasia, I used 10 hypothetical, typical patients to assess the testing attitudes of 33 experienced colonoscopists. There was great disagreement on the type and frequency of the advised diagnostic investigation in some cases.
openaire   +2 more sources

Colorectal Neuroendocrine Neoplasms

Neuroendocrinology, 2019
The subject of colorectal neuroendocrine neoplasms (NENs), subdivided into well-differentiated NENs, termed neuroendocrine tumours (NETs; grade (G) 1 and 2), and poorly differentiated NENs, termed neuroendocrine carcinomas (NECs; G3) according to the 2010 World Health Organisation (WHO) classification, has arguably not had as much attention or study as
Ramage, John K, Valle, Juan W, Nieveen van Dijkum, Els J M, Sundin, Anders, Pascher, Andreas, Couvelard, Anne, Kloeppel, Guenter   +6 more
openaire   +1 more source

Origins and morphogenesis of colorectal neoplasms

APMIS, 1999
Gastrointestinal stem cells are considered pivotal in colonic carcinogenesis. There is evidence to suggest that early microadenomas in the colon are polyclonal in origin. Adenomas, once initiated, enlarge by the process of crypt fission. It is also the main mechanism by which neoplastic clones spread through the colorectal epithelium. Both concepts are
W M, Wong, S B, Garcia, N A, Wright
openaire   +2 more sources

Benign colorectal neoplasms undetected by colonoscopy

Gastrointestinal Radiology, 1992
This retrospective investigation assessed the sensitivity of colonoscopy for the detection of colonic polyps seen previously at barium enema examination. Included in the study were 77 patients with 106 polypoid lesions. Films showing lesions not subsequently seen at colonoscopy were reviewed and only those lesions with a visible point of attachment ...
D W, Gelfand, M Y, Chen, D J, Ott
openaire   +2 more sources

Screening for Colorectal Neoplasms

2009
Evidence is mounting that colorectal adenocarcinoma can be prevented by detecting and removing adenomatous polyps, and that detecting early-stage cancers reduces mortality from the disease. Both polyps and early-stage cancers are usually asymptomatic; cancers that have grown large enough to cause symptoms have a much worse prognosis.
David E. Beck, Patricia L. Roberts, John L. Rombeau, Michael J. Stamos, Steven D. Wexner   +4 more
openaire   +1 more source

Multiple Colorectal Neoplasms in X-Linked Agammaglobulinemia

Clinical Gastroenterology and Hepatology, 2008
X-linked agammaglobulinemia (XLA) is a primary immunodeficiency disorder caused by germline mutation of the Bruton tyrosine kinase (BTK) gene. It is characterized by disturbed B-cell development, decreased immunoglobulin levels, and increased patient susceptibility to infection.
Brosens, Lodewijk A. A., Tytgat, Kristien M. A. J., Morsink, Folkert H. M., Sinke, Richard J., ten Berge, Ineke J. M., Giardiello, Francis M., Offerhaus, G. Johan A., Keller, Josbert J.   +7 more
openaire   +3 more sources

Chemoprevention of Colorectal Neoplasms

Archives of Surgery, 1986
The organospecific, 1,2-dimethylhydrazine-induced rat tumor model was used to test tumor formation in groups of animals receiving regular chow, powdered chow with 7%/wt ascorbic acid supplement, pelleted chow with 1%/wt beta-carotene supplement, and pelleted chow with placebo beadlets. Following a 16-week induction period, animals were killed and tumor
T A, Colacchio, V A, Memoli
openaire   +2 more sources