Results 1 to 10 of about 640 (78)
Soluble gC1qR is an autocrine signal that induces B1R expression on endothelial cells [PDF]
Bradykinin (BK) is one of the most potent vasodilator agonists known and belongs to the kinin family of proinflammatory peptides. BK induces its activity via two G protein-coupled receptors: BK receptor 1 (B1R) and BK receptor 2.
Ramadass, M +10 more
core +1 more source
Spirochetes belonging to the Borrelia (B.) burgdorferi sensu lato complex differ in their resistance to complement-mediated killing, particularly in regard to human serum.
Christine Skerka +24 more
core +1 more source
Levels in serum of components of complement were studied in a group of 10 patients with mixed cryoglobulinemia. The profiles found in most patients showed decreased levels of the early complement components C1, C4, and C2, with normal levels of C3 ...
de Rojas, Marta Patarroyo +2 more
core +1 more source
Is there a correlation with the activity of C1-esterase-inhibitor ?
Titel und Inhaltsverzeichnis 1\. Einleitung 2\. Methodik 3\. Ergebnisteil 4\. Diskussion 5\. Zusammenfassung 7\. LiteraturverzeichnisEinleitung: In dieser Studie wurde der Abfall der monozytären HLA-DR- Expression als Zeichen der ...
Türke, Ulrich
core +1 more source
Complement Factor H: solution structures and interactions with ligands
Factor H (FH) is a plasma glycoprotein that plays a central role in regulating the alternative pathway of complement. It is composed of 20 short complement regulator (SCR) domains.
Okemefuna, A.I.
core
Complement biosynthesis by the human hepatoma-derived cell line HepG2.
The human hepatoma-derived cell line, HepG2, synthesized and secreted functional complement proteins C1r, C1s, C2, C3, C4, C5, factor B, C1 inhibitor, C3b inactivator, a small amount of C6, and trace amounts of C8; but failed to produce detectable C1q,
Morris, K M +3 more
core
A Case of Duodenal Edema-related Undiagnosed Hereditary Angioedema. [PDF]
Kawara F +3 more
europepmc +1 more source
Presentation of an extraordinary colic: abdominal pain as the first and only utterance of an acquired C1-inhibitor deficiency. [PDF]
Hanevelt J +1 more
europepmc +1 more source

