Results 121 to 130 of about 6,275 (172)

Studies of α',β'-Epoxyketone Synthesis by Small-Molecule Flavins and Flavoenzymes. [PDF]

Angew Chem Int Ed Engl
Walter A, Kuttenlochner W, Eisenreich W, Yao C, Groll M, Storch G.   +5 more
europepmc   +1 more source

Correction: Complement activity and autophagy are dysregulated in the lungs of patients with nonresolvable COVID‑19 requiring lung transplantation. [PDF]

Respir Res
Shivshankar P, Mueller-Ortiz SL, Domozhirov AY, Bi W, Collum SD, Doursout MF, Patel M, LeFebvre IN, Akkanti B, Yau S, Huang HJ, Hussain R, Karmouty-Quintana H.   +12 more
europepmc   +1 more source

Altered Neuroinflammatory Transcriptomic Profile in the Hippocampal Dentate Gyrus Three Weeks After Lateral Fluid Percussion Injury in Rats. [PDF]

Int J Mol Sci
DeSana AJ, Alfawares Y, Khatri R, Hopkins TM, Best FV, McGuire JL, Ngwenya LB.   +6 more
europepmc   +1 more source

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COMPLEMENT ALLOTYPING IN SLE: ASSOCIATION WITH C4A NULL

Australian and New Zealand Journal of Medicine, 1983
Abstract: Immunogenetic factors are important in systemic lupus erythematosus (SLE) and deficiency of a number of complement components is often associated with a lupuslike illness. The complement components Bf, C2 and C4 are encoded within the human major histocompatibility complex (MHC) and are polymorphic.
F T, Christiansen, R L, Dawkins, G, Uko, J, McCluskey, P H, Kay, P J, Zilko   +5 more
openaire   +2 more sources

Complement C4A, C4B and BF haplotypes in Koreans

Tissue Antigens, 1996
Specific alleles at C4A, C4B and BF loci occur in populations and are inherited in complotypes, which are linked with particular HLA haplotypes. Considerable differences in complement allele and complotype frequencies have been observed among various ethnic groups.
K S, Park, M H, Park, T, Juji, K, Tokunaga   +3 more
openaire   +2 more sources

The Phenotype of SLE Associated with Complete Deficiency of Complement Isotype C4A

Journal of Clinical Immunology, 1998
Complete deficiency of the complement C4A isotype is a known genetic risk factor for systemic lupus erythematosus (SLE). The disease phenotype of C4A-deficient patients has never been defined. Among 200 patients with SLE from five centers, 18 (9%) with C4A deficiency were identified.
T R, Welch, C, Brickman, N, Bishof, S, Maringhini, M, Rutkowski, M, Frenzke, N, Kantor   +6 more
openaire   +2 more sources

Complement Activation Products C3a and C4a as Endogenous Antimicrobial Peptides

International Journal of Peptide Research and Therapeutics, 2009
All vertebrate species are constantly challenged by infectious agents and pathogens. In order to fight these infectious agents the human host has developed a sophisticated and powerful immune defense. The complement system, which represents the first defense line of innate immunity is activated immediately, within seconds.
Peter F. Zipfel, Michael Reuter
openaire   +1 more source

Insights into the Antibacterial Properties of Complement Peptides C3a, C4a, and C5a across Vertebrates

The Journal of Immunology, 2022
Abstract Complement peptides C3a, C4a, and C5a are important components of innate immunity in vertebrates. Although they diverged from a common ancestor, only C3a and C4a can act as antibacterial peptides in Homo sapiens, suggesting that C5a has evolved into a purely chemotactic molecule; however, the antibacterial properties of C3a, C4a,
Xu-Jie Zhang, Ya-Qin Zhong, Zi-You Ma, Ya-Zhen Hu, Jian-Guo Su, Yong-An Zhang   +5 more
openaire   +3 more sources

Glomerular deposition of the complement C4 isotypes C4A and C4B in glomerulonephritis

Nephrology Dialysis Transplantation, 1996
Complement C4 is a component of the classical complement pathway, which is a major mediator of inflammation in many forms of glomerulonephritis. The two isoforms of C4-C4A and C4B-differ in their physicochemical and functional properties.The glomerular deposition of C4A and C4B was investigated in 39 cases of glomerulonephritis with classical pathway ...
K, Lhotta, A, Schlogl, F, Kronenberg, M, Joannidis, P, Konig   +4 more
openaire   +2 more sources

Complement C4A and C4B Gene Copy Number Study in Alzheimer's Disease Patients

Current Alzheimer Research, 2017
Background/Objectives: Increasing evidence suggests the importance of neuroinflammation in the pathogenesis of Alzheimer’s disease (AD), which is a complex neurodegenerative disorder. Complement activation occurs in the brain of patients with AD and seems to contribute to an important local inflammatory state. Increased expression of the fourth serum
Michele, Zorzetto, Francesca, Datturi, Laura, Divizia, Cristiana, Pistono, Ilaria, Campo, Annalisa, De Silvestri, Mariaclara, Cuccia, Giovanni, Ricevuti   +7 more
openaire   +2 more sources