Results 161 to 170 of about 113,435 (208)

Rare predicted loss-of-function and damaging missense variants in CFHR5 associate with protection from age-related macular degeneration. [PDF]

Am J Hum Genet
Holleman AM, Deaton AM, Hoffing RA, Krohn L, LoGerfo P, Nioi P, Plekan ME, Akle Serrano S, Ticau S, Walshe TE, Borodovsky A, Ward LD.   +11 more
europepmc   +1 more source

Unraveling the Role of the Multifunctional Groups in the Adsorption of l-Cysteine on Rutile TiO₂(110). [PDF]

J Am Chem Soc
Blanco Garcia M, Perilli D, Daldossi C, Ugolotti A, Giordano M, Dolling DS, Wagstaffe M, Kohantorabi M, Stierle A, Di Valentin C, Noei H.   +10 more
europepmc   +1 more source

Identification and validation of a tear fluid-derived protein biomarker signature in patients with amyotrophic lateral sclerosis. [PDF]

Acta Neuropathol Commun
Scholl LS, Demleitner AF, Riedel J, Adachi S, Neuenroth L, Meijs C, Tzeplaeff L, Caldi Gomes L, Galhoz A, Cordts I, Lenz C, Menden M, Lingor P.   +12 more
europepmc   +1 more source

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Regulatory proteins for the activated third and fourth components of complement (C3b and C4b) in mice. I. Isolation and characterization of factor H: the serum cofactor for the C3b/C4b inactivator (Factor I)

Journal of Immunological Methods, 1984
Factor H, purified from mouse EDTA-plasma using a 4-step procedure, consists of a single polypeptide chain of Mr 150,000 on SDS-PAGE. Mouse H (Hmo) was required for the cleavage of fluid-phase mouse C3b by mouse I (Imo). The final product of degradation of fluid-phase mouse C3b was iC3b, consisting of fragments of the alpha'-chain (alpha'-70, alpha'-43)
T. Kinoshita, V. Nussenzweig
exaly   +4 more sources

Cyanate as an inactivator of complement proteins.

The Journal of Immunology, 1975
Sodium cyanate added to normal human serum or serum from patients with sickle-cell disease resulted in the functional inactivation of C3, C5, C6, C7, and the C3b inactivator, but not C8 and C9.
D. Schultz, P. I. Arnold
semanticscholar   +3 more sources

cDNA cloning, sequencing and chromosomal assignment of the gene for mouse complement factor I (C3bC4b inactivator): Identification of a species specific divergent segment in factor I

Molecular Immunology, 1996
J. Minta, M. Wong, C. Kozak, L. M. Kunnath-Muglia, G. Goldberger   +4 more
exaly   +2 more sources

Complement inactivating proteins and intraspecies venom variation in Crotalus oreganus helleri

Toxicon, 2007
Complement inactivating properties were detected in venom from the southern California distribution of Crotalus oreganus helleri (Southern Pacific Rattlesnake). This activity showed strong geographic bias to the San Bernardino Mountain range, and venom from this area reacted strongly with Fraction 5 antiserum (AF5). However, venoms from the San Jacinto
Jennifer D, Jurado, Eppie D, Rael, Carl S, Lieb, Ernesto, Nakayasu, William K, Hayes, Sean P, Bush, Jeremy A, Ross   +6 more
openaire   +2 more sources

Antibody-complement-mediated cytotoxicity is enhanced by ribosome-inactivating proteins.

The Journal of Immunology, 1985
Abstract The antibody-mediated cytotoxicity of complement can be increased by the ribosome-inactivating proteins gelonin and PAP-S. Treatment of human lymphoid cells that express CALLA with an anti-CALLA monoclonal antibody, J5, and then with rabbit complement, leaves about 6% of the cells alive.
V S, Goldmacher, J, Anderson, W A, Blättler, J M, Lambert, P D, Senter   +4 more
openaire   +2 more sources

Interaction of vaccinia virus complement control protein with human complement proteins: Factor I-mediated degradation of C3b to iC3b1 inactivates the alternative complement pathway

Molecular Immunology, 1998
Abstract Vaccinia virus complement control protein (VCP) is a virulence determinant of vaccinia virus that helps protect the virus from the complement attack of the host. To characterize the interaction of VCP with C3 and C4 and understand the mechanism by which VCP inactivates complement, we have expressed VCP in a yeast expression ...
A, Sahu, S N, Isaacs, A M, Soulika, J D, Lambris   +3 more
openaire   +2 more sources

Preferential inactivation of the C5 convertase of the alternative complement pathway by factor I and membrane cofactor protein (MCP)

Molecular Immunology, 1991
Human C3b bound to the ghost of sheep erythrocytes (E*) via activation of the alternative complement pathway (E*AC3b) consists of four major constituents on SDS-PAGE of 350, 260, 210 and 180 kDa. 350 kDa C3b is a dimeric form of C3b in which the alpha' chain of one C3b binds covalently to that of the other C3b.
T, Seya, M, Okada, M, Matsumoto, K S, Hong, T, Kinoshita, J P, Atkinson   +5 more
openaire   +2 more sources