Results 131 to 140 of about 1,351 (168)

Coxsackievirus B Epidemic at a Boys' Camp

Archives of Pediatrics & Adolescent Medicine, 1979
An epidemic of coxsackievirus B2 infections occured at a boys summer camp. The resulting illness was characterized by malaise, headache, muscle pain, and high fever that persisted for four to six days. The boy in the index case arrived at the camp the first day of the season. The attack rate was 89% among campers and 47% among counselors. The spread of
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Group B Coxsackievirus Diseases

2010
Two groups of coxsackieviruses, A and B, were subsequently defined according to their pathogenicity in suckling mice: the group A coxsackieviruses caused generalized myositis, while the coxsackieviruses group B (CVB) resulted in multiorgan infections. This chapter focuses on CVB, because this group includes major human pathogens and because extensive ...
Steven Tracy, Nora M. Chapman
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Pathogenesis of Coxsackievirus B Infections

2014
Regarding the pathogenesis of enteroviral heart disease there has been uncertainty whether viral cytotoxicity or immune-mediated processes are crucial for organ pathology during acute and persistent heart muscle infection. This chapter provides experimental evidence for the decisive role of virus replication in the induction and maintenance of chronic ...
Reinhard Kandolf, Hans-Christoph Selinka, Karin Klingel   +2 more
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Pediatric Group B Coxsackievirus Infections

2008
The CVB have long been recognized as significant pathogens of infants and children. Although the major route for transmission of the CVB is fecal-oral, vertical transmission from mother to infant is also possible. This review will focus on the more common or clinically relevant CVB-related syndromes, their diagnosis, treatment, and prevention.
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A cluster of cases of neonatal coxsackievirus B meningitis and myocarditis

Journal of Paediatrics and Child Health, 1998
Three babies were referred with enteroviral meningitis and myocarditis during a 4‐month period. During this same period there was an increased frequency of coxsackievirus B type 2 isolates reported to the National Centre for Disease Control. Myocarditis was simultaneous with meningitis in one baby, but delayed by 10 weeks in another, in whom ...
A J, Daley, D, Isaacs, D E, Dwyer, G L, Gilbert   +3 more
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Roles of T Cells in Coxsackievirus B-Induced Disease

1997
Enteroviral infection, for example by coxsackieviruses, is thought to be the initiating event of human myocarditis. This is supported by epidemiological studies showing an increased incidence of viral myocarditis following endemics of coxsackie B virus (CVB) infections (Helin et al. 1968). However, the assumed pathogenetic role of enteroviral infection
P L, Schwimmbeck, S A, Huber, H P, Schultheiss   +2 more
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Perinatal Echovirus and Croup B Coxsackievirus infections

Clinics in Perinatology, 1988
Enteroviral infections late in pregnancy are common, especially during periods of high prevalence of community infection. Most of these infections, however, are not associated with significant maternal or neonatal disease. Conversely, as many as 65 per cent of women who give birth to infants with proven enteroviral infection have symptomatic disease ...
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Genetics of coxsackievirus B cardiovirulence and inflammatory heart muscle disease

Trends in Microbiology, 1996
Coxsackieviruses B (CVBs) are etiological agents of human inflammatory myocardial disease. The genetics of the coxsackieviral virulence phenotype in mice are now beginning to be understood with the availability of infectious cDNA copies of CVB genomes. Investigations to date with CVB3 and CVB4 have shown that sites within a non-translated region and in
S, Tracy, N M, Chapman, J, Romero, A I, Ramsingh   +3 more
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Purification of the Putative Coxsackievirus B Receptor from HeLa Cells

Biochemical and Biophysical Research Communications, 1997
We have identified a protein expressed by human and murine cells susceptible to coxsackievirus B3 (CVB3) infection and purified it from HeLa cells. This protein of approximately 45,000 Mr is expressed by HeLa cells and mouse fetal heart fibroblasts (susceptible to infection), and not by C3H murine fibroblasts or the human RD cell line (resistant).
S D, Carson, N N, Chapman, S M, Tracy
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Coxsackievirus B RNA Replication: Lessons from Poliovirus

2008
The replication of coxsackievirus RNA occurs with rapid onset, starting approximately 2.5 h after infection. The mechanisms entailing the RNA replication of enteroviruses, like coxsackievirus and poliovirus, are highly conserved. These processes require two steps of RNA amplification: (i) complete synthesis of the negative-strand RNA using input RNA as
P, Sean, B L, Semler
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